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Experimental Neurology|September 1, 1994
Reduction of neurological damage by a peptide segment of the amyloid beta/A4 protein precursor in a rabbit spinal cord ischemia modelM P Bowes, E Masliah, D A Otero, et al.Journal of Neuroscience Research|June 1, 1991
Expression of protein kinase C isozymes in primary neuronal cultures of the rat cerebellumS Shimohama, Y Uehara-Kunugi, K Terai, et al.Brain Research|June 14, 1988
Decreased levels of protein kinase C in Alzheimer brainG Cole, K R Dobkins, L A Hansen, et al.The American Journal of Pathology|February 1, 1992
Casein kinase II alteration precedes tau accumulation in tangle formationE Masliah, D S Iimoto, M Mallory, et al.Neuroscience Letters|November 21, 1994
Apolipoprotein-E epsilon-4 is associated with increased neurofibrillary pathology in the Lewy body variant of Alzheimer's diseaseL A Hansen, D Galasko, W Samuel, et al.Retina (Philadelphia, Pa.)|March 21, 1998
The improved image of indocyanine green angiography in young healthy volunteersS Yoneya, Y Komatsu, K Mori, et al.Brain Research|October 29, 1990
Increased immunoreactivity of brain spectrin in Alzheimer disease: a marker for synapse loss?E Masliah, D S Iimoto, T Saitoh, et al.Archives of Neurology|June 27, 2000
Association between severe cerebral amyloid angiopathy and cerebrovascular lesions in Alzheimer disease is not a spurious one attributable to apolipoprotein E4J M Olichney, L A Hansen, C R Hofstetter, et al.Annals of the New York Academy of Sciences|May 20, 2000
Relationship between severe amyloid angiopathy, apolipoprotein E genotype, and vascular lesions in Alzheimer's diseaseJ M Olichney, L A Hansen, J H Lee, et al.Brain and Cognition|July 1, 1996
Neuropsychological deficits associated with diffuse Lewy body diseaseD P Salmon, D Galasko, L A Hansen, et al.Pageof 51