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Plos One|July 25, 2015
Gene Expression in Experimental Aortic Coarctation and Repair: Candidate Genes for Therapeutic Intervention?John F LaDisa, Serdar Bozdag, Jessica Olson, et al.
Cardiovascular Research|May 1, 1997
Temporal dependence of coronary collateral developmentJ R Kersten, M F McGough, P S Pagel, et al.
The American Journal of Physiology|July 31, 1998
Acute hyperglycemia abolishes ischemic preconditioning in vivoJ R Kersten, T J Schmeling, K G Orth, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 6, 2000
Diabetes abolishes ischemic preconditioning: role of glucose, insulin, and osmolalityJ R Kersten, W G Toller, E R Gross, et al.
American Journal of Physiology. Heart and Circulatory Physiology|October 23, 2001
Hyperglycemia reduces coronary collateral blood flow through a nitric oxide-mediated mechanismJ R Kersten, W G Toller, J P Tessmer, et al.
Annals of Biomedical Engineering|April 13, 1999
Changes in passive but not active mechanical properties predict recovery of function of stunned myocardiumT J Schmeling, D A Hettrick, J R Kersten, et al.
American Journal of Physiology. Heart and Circulatory Physiology|March 27, 2004
Cardioprotection by glucose-insulin-potassium: dependence on KATP channel opening and blood glucose concentration before ischemiaJohn F LaDisa, John G Krolikowski, Paul S Pagel, et al.
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