Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

R Kofler

Showing results (61-70 of 209) with videos related to

Pageof 21
Sort By:
The Journal of Clinical Endocrinology and Metabolism|February 1, 1995
Amino acid substitution R384P in aldosterone synthase causes corticosterone methyloxidase type I deficiencyS Geley, K Jöhrer, M Peter, et al.
Cancer Research|November 1, 1996
Resistance to glucocorticoid-induced apoptosis in human T-cell acute lymphoblastic leukemia CEM-C1 cells is due to insufficient glucocorticoid receptor expressionS Geley, B L Hartmann, M Hala, et al.
Human Mutation|July 17, 1999
A rapid screening for steroid 21-hydroxylase mutations in patients with congenital adrenal hyperplasia. Mutations in brief no. 247. OnlineK Kapelari, Z Ghanaati, H Wollmann, et al.
British Journal of Haematology|September 13, 2001
Increased lactate production follows loss of mitochondrial membrane potential during apoptosis of human leukaemia cellsM Tiefenthaler, A Amberger, N Bacher, et al.
The Journal of Biological Chemistry|March 30, 2001
The cell cycle inhibitor p16(INK4A) sensitizes lymphoblastic leukemia cells to apoptosis by physiologic glucocorticoid levelsM J Ausserlechner, P Obexer, G J Wiegers, et al.
Wiener Klinische Wochenschrift|January 1, 1991
[Clinical epidemiologic data of malignant melanoma based on 1,368 patients of the Graz University Dermatology Clinic (1970-1989)]E Rieger, H P Soyer, R Kofler, et al.
The Journal of Biological Chemistry|July 10, 2001
The cell cycle inhibitor p16(INK4A) sensitizes lymphoblastic leukemia cells to apoptosis by physiologic glucocorticoid levelsM J Ausserlechner, P Obexer, G J Wiegers, et al.
The Journal of Experimental Medicine|March 1, 1986
Delineation of a defect in T cell receptor beta genes of NZW mice predisposed to autoimmunityD J Noonan, R Kofler, P A Singer, et al.
Cancer Research|January 1, 1997
Bcl-2 and Bcl-XL antagonize the mitochondrial dysfunction preceding nuclear apoptosis induced by chemotherapeutic agentsD Decaudin, S Geley, T Hirsch, et al.
Journal of Cutaneous Pathology|June 1, 1993
Comparison of proliferative activity as assessed by proliferating cell nuclear antigen (PCNA) and Ki-67 monoclonal antibodies in melanocytic skin lesions. A quantitative immunohistochemical studyE Rieger, R Hofmann-Wellenhof, H P Soyer, et al.
Pageof 21

Showing results (61-70 of 209) with videos related to

Sort By:
Pageof 21
The Journal of Clinical Endocrinology and Metabolism|February 1, 1995
Amino acid substitution R384P in aldosterone synthase causes corticosterone methyloxidase type I deficiencyS Geley, K Jöhrer, M Peter, et al.
Cancer Research|November 1, 1996
Resistance to glucocorticoid-induced apoptosis in human T-cell acute lymphoblastic leukemia CEM-C1 cells is due to insufficient glucocorticoid receptor expressionS Geley, B L Hartmann, M Hala, et al.
Human Mutation|July 17, 1999
A rapid screening for steroid 21-hydroxylase mutations in patients with congenital adrenal hyperplasia. Mutations in brief no. 247. OnlineK Kapelari, Z Ghanaati, H Wollmann, et al.
British Journal of Haematology|September 13, 2001
Increased lactate production follows loss of mitochondrial membrane potential during apoptosis of human leukaemia cellsM Tiefenthaler, A Amberger, N Bacher, et al.
The Journal of Biological Chemistry|March 30, 2001
The cell cycle inhibitor p16(INK4A) sensitizes lymphoblastic leukemia cells to apoptosis by physiologic glucocorticoid levelsM J Ausserlechner, P Obexer, G J Wiegers, et al.
Wiener Klinische Wochenschrift|January 1, 1991
[Clinical epidemiologic data of malignant melanoma based on 1,368 patients of the Graz University Dermatology Clinic (1970-1989)]E Rieger, H P Soyer, R Kofler, et al.
The Journal of Biological Chemistry|July 10, 2001
The cell cycle inhibitor p16(INK4A) sensitizes lymphoblastic leukemia cells to apoptosis by physiologic glucocorticoid levelsM J Ausserlechner, P Obexer, G J Wiegers, et al.
The Journal of Experimental Medicine|March 1, 1986
Delineation of a defect in T cell receptor beta genes of NZW mice predisposed to autoimmunityD J Noonan, R Kofler, P A Singer, et al.
Cancer Research|January 1, 1997
Bcl-2 and Bcl-XL antagonize the mitochondrial dysfunction preceding nuclear apoptosis induced by chemotherapeutic agentsD Decaudin, S Geley, T Hirsch, et al.
Journal of Cutaneous Pathology|June 1, 1993
Comparison of proliferative activity as assessed by proliferating cell nuclear antigen (PCNA) and Ki-67 monoclonal antibodies in melanocytic skin lesions. A quantitative immunohistochemical studyE Rieger, R Hofmann-Wellenhof, H P Soyer, et al.
Pageof 21