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R L Neve

Showing results (121-130 of 136) with videos related to

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Molecular and Cellular Neurosciences|May 19, 2001
Beta-amyloid peptide expression is sufficient for myotube death: implications for human inclusion body myopathyH W Querfurth, T Suhara, K M Rosen, et al.
Journal of Neurochemistry|August 11, 2000
Overexpression of HuD, but not of its truncated form HuD I+II, promotes GAP-43 gene expression and neurite outgrowth in PC12 cells in the absence of nerve growth factorK D Anderson, M A Morin, A Beckel-Mitchener, et al.
American Journal of Medical Genetics|May 1, 1989
Quantitative calibration and use of DNA probes for investigating chromosome abnormalities in the Prader-Willi syndromeU Tantravahi, R D Nicholls, H Stroh, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 1993
MEF2C, a MADS/MEF2-family transcription factor expressed in a laminar distribution in cerebral cortexD Leifer, D Krainc, Y T Yu, et al.
The Journal of Biological Chemistry|October 6, 1997
Neuronal expression of beta-amyloid precursor protein Alzheimer mutations causes intracellular accumulation of a C-terminal fragment containing both the amyloid beta and cytoplasmic domainsD L McPhie, R K Lee, C B Eckman, et al.
Science (New York, N.Y.)|December 18, 1998
Regulation of cocaine reward by CREBW A Carlezon, J Thome, V G Olson, et al.
Cell|May 5, 2001
EphA receptors regulate growth cone dynamics through the novel guanine nucleotide exchange factor ephexinS M Shamah, M Z Lin, J L Goldberg, et al.
Molecular Psychiatry|May 31, 2017
The dendritic spine morphogenic effects of repeated cocaine use occur through the regulation of serum response factor signalingM E Cahill, D M Walker, A M Gancarz, et al.
American Journal of Medical Genetics|July 1, 1989
Characterization of an unusual and complex chromosome 21 rearrangement using somatic cell genetics and cloned DNA probesM L Van Keuren, G D Stewart, C M Bradley, et al.
Science (New York, N.Y.)|October 30, 1987
Absence of duplication of chromosome 21 genes in familial and sporadic Alzheimer's diseaseP H St George-Hyslop, R E Tanzi, R J Polinsky, et al.
Pageof 14

Showing results (121-130 of 136) with videos related to

Sort By:
Pageof 14
Molecular and Cellular Neurosciences|May 19, 2001
Beta-amyloid peptide expression is sufficient for myotube death: implications for human inclusion body myopathyH W Querfurth, T Suhara, K M Rosen, et al.
Journal of Neurochemistry|August 11, 2000
Overexpression of HuD, but not of its truncated form HuD I+II, promotes GAP-43 gene expression and neurite outgrowth in PC12 cells in the absence of nerve growth factorK D Anderson, M A Morin, A Beckel-Mitchener, et al.
American Journal of Medical Genetics|May 1, 1989
Quantitative calibration and use of DNA probes for investigating chromosome abnormalities in the Prader-Willi syndromeU Tantravahi, R D Nicholls, H Stroh, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 15, 1993
MEF2C, a MADS/MEF2-family transcription factor expressed in a laminar distribution in cerebral cortexD Leifer, D Krainc, Y T Yu, et al.
The Journal of Biological Chemistry|October 6, 1997
Neuronal expression of beta-amyloid precursor protein Alzheimer mutations causes intracellular accumulation of a C-terminal fragment containing both the amyloid beta and cytoplasmic domainsD L McPhie, R K Lee, C B Eckman, et al.
Science (New York, N.Y.)|December 18, 1998
Regulation of cocaine reward by CREBW A Carlezon, J Thome, V G Olson, et al.
Cell|May 5, 2001
EphA receptors regulate growth cone dynamics through the novel guanine nucleotide exchange factor ephexinS M Shamah, M Z Lin, J L Goldberg, et al.
Molecular Psychiatry|May 31, 2017
The dendritic spine morphogenic effects of repeated cocaine use occur through the regulation of serum response factor signalingM E Cahill, D M Walker, A M Gancarz, et al.
American Journal of Medical Genetics|July 1, 1989
Characterization of an unusual and complex chromosome 21 rearrangement using somatic cell genetics and cloned DNA probesM L Van Keuren, G D Stewart, C M Bradley, et al.
Science (New York, N.Y.)|October 30, 1987
Absence of duplication of chromosome 21 genes in familial and sporadic Alzheimer's diseaseP H St George-Hyslop, R E Tanzi, R J Polinsky, et al.
Pageof 14