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Journal of Immunology (Baltimore, Md. : 1950)|March 1, 1982
Human eosinophil lysophospholipase: the sole protein component of Charcot-Leyden crystalsP F Weller, D Bach, K F AustenJournal of Immunology (Baltimore, Md. : 1950)|March 1, 1975
Inhibition by epsilon-aminocaproic acid of the activation of the first component of the complement systemN A Soter, K F Austen, I GigliArthritis and Rheumatism|July 1, 1975
Depressed synovial fluid levels of properdin and properdin factor B in patients with rheumatoid arthritisS Ruddy, D T Fearon, K F AustenThe Journal of Biological Chemistry|September 5, 1989
Identification of an aldehyde dehydrogenase in the microsomes of human polymorphonuclear leukocytes that metabolizes 20-aldehyde leukotriene B4J Sutyak, K F Austen, R J SobermanThe Journal of Experimental Medicine|September 1, 1972
Immunological release of histamine and slow reacting substance of anaphylaxis from human lungM Kaliner, R P Orange, K F AustenThe Journal of Experimental Medicine|December 1, 1971
Modulation of function of the activated first component of complement by a fragment derived from serum. I. Effect on early components of complementI Gigli, A P Kaplan, K F AustenThe American Journal of Pathology|April 1, 1992
Mast-cell phenotype in indolent forms of mastocytosis. Ultrastructural features, fluorescence detection of avidin binding, and immunofluorescent determination of chymase, tryptase, and carboxypeptidaseN Weidner, R F Horan, K F AustenThe Journal of Clinical Investigation|June 11, 1991
Eosinophils altered phenotypically and primed by culture with granulocyte/macrophage colony-stimulating factor and 3T3 fibroblasts generate leukotriene C4 in response to FMLPW F Owen, J Petersen, K F AustenJournal of Immunology (Baltimore, Md. : 1950)|January 1, 1981
Isolation of C4-binding protein from guinea pig plasma and demonstration of its function as a control protein of the classical complement pathway C3 convertaseJ Burge, A Nicholson-Weller, K F AustenThe Journal of Experimental Medicine|December 1, 1973
Formation of a hemolytically active cellular intermediate by the interaction between properdin factors B and D and the activated third component of complementD T Fearon, K F Austen, S RuddyPageof 49