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Circulation|August 29, 2001
Reduced sarco/endoplasmic reticulum Ca(2+) uptake activity can account for the reduced response to NO, but not sodium nitroprusside, in hypercholesterolemic rabbit aortaT Adachi, R Matsui, R M Weisbrod, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|February 1, 1997
Reduced responsiveness of hypercholesterolemic rabbit aortic smooth muscle cells to nitric oxideR M Weisbrod, M C Griswold, Y Du, et al.
Circulation Research|February 5, 1999
Mechanism of nitric oxide-induced vasodilatation: refilling of intracellular stores by sarcoplasmic reticulum Ca2+ ATPase and inhibition of store-operated Ca2+ influxR A Cohen, R M Weisbrod, M Gericke, et al.
The American Journal of Physiology|September 11, 1996
Vasodilatory properties of recombinant maxadilanT S Jackson, E Lerner, R M Weisbrod, et al.
The Journal of Biological Chemistry|December 13, 2000
Properties of a native cation channel activated by Ca2+ store depletion in vascular smooth muscle cellsE S Trepakova, M Gericke, Y Hirakawa, et al.
British Journal of Pharmacology|January 14, 1999
Evidence that additional mechanisms to cyclic GMP mediate the decrease in intracellular calcium and relaxation of rabbit aortic smooth muscle to nitric oxideR M Weisbrod, M C Griswold, M Yaghoubi, et al.
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