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Circulation|April 3, 2001
Combined and individual mitochondrial HSP60 and HSP10 expression in cardiac myocytes protects mitochondrial function and prevents apoptotic cell deaths induced by simulated ischemia-reoxygenationK M Lin, B Lin, I Y Lian, et al.Circulation|September 15, 1996
Overexpression of heat shock protein 72 in transgenic mice decreases infarct size in vivoJ J Hutter, R Mestril, E K Tam, et al.The Journal of Clinical Investigation|April 1, 1995
Overexpression of the rat inducible 70-kD heat stress protein in a transgenic mouse increases the resistance of the heart to ischemic injuryM S Marber, R Mestril, S H Chi, et al.The Journal of Clinical Investigation|March 21, 1998
Protection against myocardial dysfunction after a brief ischemic period in transgenic mice expressing inducible heat shock protein 70S U Trost, J H Omens, W J Karlon, et al.Physiological Genomics|November 14, 2000
Functional properties of skeletal muscle from transgenic animals with upregulated heat shock protein 70T M Nosek, M A Brotto, D A Essig, et al.Human Molecular Genetics|July 13, 2001
Over-expression of inducible HSP70 chaperone suppresses neuropathology and improves motor function in SCA1 miceC J Cummings, Y Sun, P Opal, et al.Metabolism: Clinical and Experimental|March 1, 1982
Influence of thyroid hormone administration on myosin ATPase activity and myosin isoenzyme distribution in the heart of diabetic ratsW H DillmannAnnual Review of Medicine|January 1, 1989
Diabetes and thyroid-hormone-induced changes in cardiac function and their molecular basisW H DillmannAnnals of the New York Academy of Sciences|December 22, 1999
Influences of increased expression of the Ca2+ ATPase of the sarcoplasmic reticulum by a transgenic approach on cardiac contractilityW H DillmannInfectious Diseases in Obstetrics and Gynecology|May 7, 1999
Heat shock proteins and protection against ischemic injuryW H DillmannPageof 11