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The Journal of General Physiology|April 18, 1998
Effect of bay K 8644 (-) and the beta2a subunit on Ca2+-dependent inactivation in alpha1C Ca2+ channelsF Noceti, R Olcese, N Qin, et al.
The Journal of General Physiology|February 7, 2001
A conducting state with properties of a slow inactivated state in a shaker K(+) channel mutantR Olcese, D Sigg, R Latorre, et al.
The Journal of Membrane Biology|August 13, 2002
External pore collapse as an inactivation mechanism for Kv4.3 K+ channelsM Eghbali, R Olcese, M M Zarei, et al.
The Journal of General Physiology|June 20, 1998
Fast inactivation in Shaker K+ channels. Properties of ionic and gating currentsM J Roux, R Olcese, L Toro, et al.
Biophysical Journal|May 29, 2000
Expression of the alpha(2)delta subunit interferes with prepulse facilitation in cardiac L-type calcium channelsD Platano, N Qin, F Noceti, et al.
FEBS Letters|November 11, 1999
Hormonal control of protein expression and mRNA levels of the MaxiK channel alpha subunit in myometriumM Song, N Zhu, R Olcese, et al.
The Journal of Physiology|December 15, 1996
Coupling between charge movement and pore opening in vertebrate neuronal alpha 1E calcium channelsR Olcese, A Neely, N Qin, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 18, 1997
Feedback inhibition of Ca2+ channels by Ca2+ depends on a short sequence of the C terminus that does not include the Ca2+ -binding function of a motif with similarity to Ca2+ -binding domainsJ Zhou, R Olcese, N Qin, et al.
Receptors & Channels|January 1, 1996
Increase in Ca2+ channel expression by deletions at the amino terminus of the cardiac alpha 1C subunitX Wei, A Neely, R Olcese, et al.
The American Journal of Physiology|March 1, 1995
Dual activation of the cardiac Ca2+ channel alpha 1C-subunit and its modulation by the beta-subunitA Neely, R Olcese, P Baldelli, et al.
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