Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

R Oner

Showing results (21-30 of 38) with videos related to

Pageof 4
Sort By:
Hemoglobin|March 18, 2000
Beta-thalassemia intermedia associated with homozygosity for the -87 (C-->T) mutation in a Turkish familyF Gümrük, H Mergen, R Oner, et al.
Hemoglobin|January 17, 2002
Severe beta-thalassemia in frameshift codon 6 (-A) homozygotes: effects of haplotype on phenotypeE Birben, C Oner, R Oner, et al.
Haematologica|March 1, 1992
Beta-globin haplotype and XmnI polymorphism at position G (gamma)-158 and HbF production in Fanconi's anemiaM C Rosatelli, C Altay, R Oner, et al.
British Journal of Haematology|October 1, 1991
Dominant beta-thalassaemia trait in a Portuguese family is caused by a deletion of (G)TGGCTGGTGT(G) and an insertion of (G)GCAG(G) in codons 134, 135, 136 and 137 of the beta-globin geneR Oner, C Oner, J B Wilson, et al.
Hemoglobin|September 7, 2000
Molecular analysis of turkish beta-thalassemia heterozygotes with normal Hb A2 levelsR Oner, E Birben, C Acar, et al.
The Turkish Journal of Pediatrics|April 1, 1997
Compound heterozygosity for hemoglobin Knossos [alpha 2 beta 2 27 (B9) Ala-Ser] and IVS I-1 mutationA Gürgey, H Balkan, G Irken, et al.
Hemoglobin|January 1, 1991
Beta-thalassemia, HB S-beta-thalassemia and sickle cell anemia among TunisiansS Fattoum, F Guemira, C Oner, et al.
American Journal of Hematology|July 30, 2004
Severe hemolytic anemia associated with Hb Volga [beta27(B9)Ala-->Asp]: GCC-->GAC at codon 27 in a Turkish familyM Sözen, C Karaaslan, R Oner, et al.
Human Genetics|January 1, 1994
The T-->C mutation at position +96 of the untranslated region 3' to the terminating codon of the beta-globin gene is a rare polymorphism that does not cause a beta-thalassemia as previously ascribedV Divoky, E Baysal, R Oner, et al.
Acta Haematologica|January 1, 1996
A novel (delta beta)(0)-thalassemia due to a approximately 30-kb deletion observed in a Turkish familyR Oner, C Oner, G Erdem, et al.
Pageof 4

Showing results (21-30 of 38) with videos related to

Sort By:
Pageof 4
Hemoglobin|March 18, 2000
Beta-thalassemia intermedia associated with homozygosity for the -87 (C-->T) mutation in a Turkish familyF Gümrük, H Mergen, R Oner, et al.
Hemoglobin|January 17, 2002
Severe beta-thalassemia in frameshift codon 6 (-A) homozygotes: effects of haplotype on phenotypeE Birben, C Oner, R Oner, et al.
Haematologica|March 1, 1992
Beta-globin haplotype and XmnI polymorphism at position G (gamma)-158 and HbF production in Fanconi's anemiaM C Rosatelli, C Altay, R Oner, et al.
British Journal of Haematology|October 1, 1991
Dominant beta-thalassaemia trait in a Portuguese family is caused by a deletion of (G)TGGCTGGTGT(G) and an insertion of (G)GCAG(G) in codons 134, 135, 136 and 137 of the beta-globin geneR Oner, C Oner, J B Wilson, et al.
Hemoglobin|September 7, 2000
Molecular analysis of turkish beta-thalassemia heterozygotes with normal Hb A2 levelsR Oner, E Birben, C Acar, et al.
The Turkish Journal of Pediatrics|April 1, 1997
Compound heterozygosity for hemoglobin Knossos [alpha 2 beta 2 27 (B9) Ala-Ser] and IVS I-1 mutationA Gürgey, H Balkan, G Irken, et al.
Hemoglobin|January 1, 1991
Beta-thalassemia, HB S-beta-thalassemia and sickle cell anemia among TunisiansS Fattoum, F Guemira, C Oner, et al.
American Journal of Hematology|July 30, 2004
Severe hemolytic anemia associated with Hb Volga [beta27(B9)Ala-->Asp]: GCC-->GAC at codon 27 in a Turkish familyM Sözen, C Karaaslan, R Oner, et al.
Human Genetics|January 1, 1994
The T-->C mutation at position +96 of the untranslated region 3' to the terminating codon of the beta-globin gene is a rare polymorphism that does not cause a beta-thalassemia as previously ascribedV Divoky, E Baysal, R Oner, et al.
Acta Haematologica|January 1, 1996
A novel (delta beta)(0)-thalassemia due to a approximately 30-kb deletion observed in a Turkish familyR Oner, C Oner, G Erdem, et al.
Pageof 4