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R P Hebbel

Showing results (61-70 of 116) with videos related to

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Biochimica Et Biophysica Acta|November 6, 1986
Inhibition of erythrocyte Ca2+-ATPase by activated oxygen through thiol- and lipid-dependent mechanismsR P Hebbel, O Shalev, W Foker, et al.
The Journal of Clinical Investigation|January 5, 2000
Origins of circulating endothelial cells and endothelial outgrowth from bloodY Lin, D J Weisdorf, A Solovey, et al.
The Journal of Clinical Investigation|June 13, 1998
Tissue factor expression by endothelial cells in sickle cell anemiaA Solovey, L Gui, N S Key, et al.
Blood|June 15, 1991
Lipid hydroperoxides permit deformation-dependent leak of monovalent cation from erythrocytesT Sugihara, W Rawicz, E A Evans, et al.
Blood|March 27, 2001
Modulation of endothelial cell activation in sickle cell disease: a pilot studyA A Solovey, A N Solovey, J Harkness, et al.
The Journal of Laboratory and Clinical Medicine|November 1, 1974
Blood cell abnormalities complicating the hypophosphatemia of hyperalimentation: erythrocyte and platelet ATP deficiency associated with hemolytic anemia and bleeding in hyperalimented dogsY Yawata, R P Hebbel, S Silvis, et al.
Angiogenesis|October 1, 2003
Binding and displacement of vascular endothelial growth factor (VEGF) by thrombospondin: effect on human microvascular endothelial cell proliferation and angiogenesisK Gupta, P Gupta, R Wild, et al.
JAMA|June 16, 1978
Extreme but asymptomatic carboxyhemoglobinemia and chronic lung diseaseR P Hebbel, J W Eaton, S Modler, et al.
The Journal of Clinical Investigation|December 1, 1982
Spontaneous oxygen radical generation by sickle erythrocytesR P Hebbel, J W Eaton, M Balasingam, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 1, 1988
Accelerated autoxidation and heme loss due to instability of sickle hemoglobinR P Hebbel, W T Morgan, J W Eaton, et al.
Pageof 12

Showing results (61-70 of 116) with videos related to

Sort By:
Pageof 12
Biochimica Et Biophysica Acta|November 6, 1986
Inhibition of erythrocyte Ca2+-ATPase by activated oxygen through thiol- and lipid-dependent mechanismsR P Hebbel, O Shalev, W Foker, et al.
The Journal of Clinical Investigation|January 5, 2000
Origins of circulating endothelial cells and endothelial outgrowth from bloodY Lin, D J Weisdorf, A Solovey, et al.
The Journal of Clinical Investigation|June 13, 1998
Tissue factor expression by endothelial cells in sickle cell anemiaA Solovey, L Gui, N S Key, et al.
Blood|June 15, 1991
Lipid hydroperoxides permit deformation-dependent leak of monovalent cation from erythrocytesT Sugihara, W Rawicz, E A Evans, et al.
Blood|March 27, 2001
Modulation of endothelial cell activation in sickle cell disease: a pilot studyA A Solovey, A N Solovey, J Harkness, et al.
The Journal of Laboratory and Clinical Medicine|November 1, 1974
Blood cell abnormalities complicating the hypophosphatemia of hyperalimentation: erythrocyte and platelet ATP deficiency associated with hemolytic anemia and bleeding in hyperalimented dogsY Yawata, R P Hebbel, S Silvis, et al.
Angiogenesis|October 1, 2003
Binding and displacement of vascular endothelial growth factor (VEGF) by thrombospondin: effect on human microvascular endothelial cell proliferation and angiogenesisK Gupta, P Gupta, R Wild, et al.
JAMA|June 16, 1978
Extreme but asymptomatic carboxyhemoglobinemia and chronic lung diseaseR P Hebbel, J W Eaton, S Modler, et al.
The Journal of Clinical Investigation|December 1, 1982
Spontaneous oxygen radical generation by sickle erythrocytesR P Hebbel, J W Eaton, M Balasingam, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 1, 1988
Accelerated autoxidation and heme loss due to instability of sickle hemoglobinR P Hebbel, W T Morgan, J W Eaton, et al.
Pageof 12