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American Journal of Clinical Pathology
|
June 1, 1986
Cardiac amyloid deposits in endomyocardial biopsies. Light microscopic, ultrastructural, and immunohistochemical studies
H Frenzel, B Schwartzkopff, H Kuhn, et al.
Der Hautarzt; Zeitschrift Fur Dermatologie, Venerologie, Und Verwandte Gebiete
|
May 1, 1993
[Amyloidosis cutis nodularis atrophicans (Gottron)]
M Widmer, E Reverdin, E Küng, et al.
Journal of Comparative Pathology
|
July 1, 1984
Immunohistochemical identification and crossreactions of amyloid-A fibril protein in man and eleven other species
R P Linke, P R Hol, E Gruys, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
April 1, 1975
Isolation of a low-molecular-weight serum component antigenically related to an amyloid fibril protein of unknown origin
R P Linke, J D Sipe, P S Pollock, et al.
Archives of Dermatology
|
November 1, 1988
Hemorrhagic bullous amyloidosis. A histologic, immunocytochemical, and ultrastructural study of two patients
T Bieber, T Ruzicka, R P Linke, et al.
Blood
|
November 1, 1975
Immunoglobulin D myeloma and amyloidosis: immunochemical and structural studies of Bence Jones and amyloid fibrillar proteins
G C White, R J Jacobson, R A Binder, et al.
Kidney International
|
October 1, 1989
Lysine-specific cleavage of beta 2-microglobulin in amyloid deposits associated with hemodialysis
R P Linke, H Hampl, H Lobeck, et al.
Archives of Dermatology
|
June 1, 1993
Fibrillar protein deposits with tubular substructure in a systemic disease beginning as cutis laxa
K M Niemi, I Anton-Lamprecht, I Virtanen, et al.
European Journal of Immunology
|
October 1, 1987
Human complement factor H: expression of an additional truncated gene product of 43 kDa in human liver
W Schwaeble, J Zwirner, T F Schulz, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
June 25, 1996
A new isoleucine substitution of Val-20 in transthyretin tetramers selectively impairs dimer-dimer contacts and causes systemic amyloidosis
D E Jenne, K Denzel, P Blätzinger, et al.
Page
of 15
Search research articles
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Showing results (111-120 of 147) with videos related to
Sort By:
Page
of 15
American Journal of Clinical Pathology
|
June 1, 1986
Cardiac amyloid deposits in endomyocardial biopsies. Light microscopic, ultrastructural, and immunohistochemical studies
H Frenzel, B Schwartzkopff, H Kuhn, et al.
Der Hautarzt; Zeitschrift Fur Dermatologie, Venerologie, Und Verwandte Gebiete
|
May 1, 1993
[Amyloidosis cutis nodularis atrophicans (Gottron)]
M Widmer, E Reverdin, E Küng, et al.
Journal of Comparative Pathology
|
July 1, 1984
Immunohistochemical identification and crossreactions of amyloid-A fibril protein in man and eleven other species
R P Linke, P R Hol, E Gruys, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
April 1, 1975
Isolation of a low-molecular-weight serum component antigenically related to an amyloid fibril protein of unknown origin
R P Linke, J D Sipe, P S Pollock, et al.
Archives of Dermatology
|
November 1, 1988
Hemorrhagic bullous amyloidosis. A histologic, immunocytochemical, and ultrastructural study of two patients
T Bieber, T Ruzicka, R P Linke, et al.
Blood
|
November 1, 1975
Immunoglobulin D myeloma and amyloidosis: immunochemical and structural studies of Bence Jones and amyloid fibrillar proteins
G C White, R J Jacobson, R A Binder, et al.
Kidney International
|
October 1, 1989
Lysine-specific cleavage of beta 2-microglobulin in amyloid deposits associated with hemodialysis
R P Linke, H Hampl, H Lobeck, et al.
Archives of Dermatology
|
June 1, 1993
Fibrillar protein deposits with tubular substructure in a systemic disease beginning as cutis laxa
K M Niemi, I Anton-Lamprecht, I Virtanen, et al.
European Journal of Immunology
|
October 1, 1987
Human complement factor H: expression of an additional truncated gene product of 43 kDa in human liver
W Schwaeble, J Zwirner, T F Schulz, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
June 25, 1996
A new isoleucine substitution of Val-20 in transthyretin tetramers selectively impairs dimer-dimer contacts and causes systemic amyloidosis
D E Jenne, K Denzel, P Blätzinger, et al.
Page
of 15