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Molecular Microbiology|August 8, 2001
Molecular genetics of SaPI1--a mobile pathogenicity island in Staphylococcus aureusA Ruzin, J Lindsay, R P NovickInfection and Immunity|April 9, 2001
Subinhibitory clindamycin differentially inhibits transcription of exoprotein genes in Staphylococcus aureusS Herbert, P Barry, R P NovickJournal of Molecular Biology|November 20, 1986
Small Staphylococcus aureus plasmids are transduced as linear multimers that are formed and resolved by replicative processesR P Novick, I Edelman, S LofdahlProceedings of the National Academy of Sciences of the United States of America|November 1, 1990
Initiation of rolling-circle replication in pT181 plasmid: initiator protein enhances cruciform extrusion at the originP Noirot, J Bargonetti, R P NovickScience (New York, N.Y.)|June 27, 1997
Bacterial interference caused by autoinducing peptide variantsG Ji, R Beavis, R P NovickJournal of Bacteriology|October 1, 1991
A temporal signal, independent of agr, is required for hla but not spa transcription in Staphylococcus aureusF Vandenesch, J Kornblum, R P NovickThe Journal of Biological Chemistry|December 6, 1996
The inactive pT181 initiator heterodimer, RepC/C, binds but fails to induce melting of the plasmid replication originR Jin, X Zhou, R P NovickMicrobes and Infection|June 22, 2001
Pathogenicity and resistance islands of staphylococciR P Novick, P Schlievert, A RuzinJournal of Bacteriology|January 1, 1997
In vitro inhibitory activity of RepC/C*, the inactivated form of the pT181 plasmid initiation protein, RepCR Jin, A Rasooly, R P NovickJournal of Bacteriology|April 1, 1994
Plasmids of the pT181 family show replication-specific initiator protein modificationA Rasooly, S J Projan, R P NovickPageof 255