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R Raghupathi

Showing results (71-80 of 80) with videos related to

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Journal of Neurotrauma|February 16, 2000
The novel compound LOE 908 attenuates acute neuromotor dysfunction but not cognitive impairment or cortical tissue loss following traumatic brain injury in ratsJ A Cheney, A L Brown, F M Bareyre, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|April 27, 2001
The maxi-K channel opener BMS-204352 attenuates regional cerebral edema and neurologic motor impairment after experimental brain injuryJ A Cheney, J D Weisser, F M Bareyre, et al.
Journal of Neurotrauma|April 1, 1997
Delayed administration of basic fibroblast growth factor (bFGF) attenuates cognitive dysfunction following parasagittal fluid percussion brain injury in the ratK L McDermott, R Raghupathi, S C Fernandez, et al.
Experimental Neurology|May 1, 1999
Genetic approaches to neurotrauma research: opportunities and potential pitfalls of murine modelsO Steward, P E Schauwecker, L Guth, et al.
Experimental Neurology|April 29, 2000
Brain trauma in aged transgenic mice induces regression of established abeta depositsY Nakagawa, L Reed, M Nakamura, et al.
Journal of Neurosurgery|November 13, 2001
Mild head injury increasing the brain's vulnerability to a second concussive impactH L Laurer, F M Bareyre, V M Lee, et al.
The Journal of Comparative Neurology|March 26, 1998
Twofold overexpression of human beta-amyloid precursor proteins in transgenic mice does not affect the neuromotor, cognitive, or neurodegenerative sequelae following experimental brain injuryH Murai, J E Pierce, R Raghupathi, et al.
The American Journal of Pathology|September 15, 1998
Brain trauma induces massive hippocampal neuron death linked to a surge in beta-amyloid levels in mice overexpressing mutant amyloid precursor proteinD H Smith, M Nakamura, T K McIntosh, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|November 10, 2001
A review and rationale for the use of genetically engineered animals in the study of traumatic brain injuryL Longhi, K E Saatman, R Raghupathi, et al.
The Journal of Comparative Neurology|July 22, 1999
Traumatic brain injury in young, amyloid-beta peptide overexpressing transgenic mice induces marked ipsilateral hippocampal atrophy and diminished Abeta deposition during agingY Nakagawa, M Nakamura, T K McIntosh, et al.
Pageof 8

Showing results (71-80 of 80) with videos related to

Sort By:
Pageof 8
You have reached the last page of results.This site can display upto 80 results.
Journal of Neurotrauma|February 16, 2000
The novel compound LOE 908 attenuates acute neuromotor dysfunction but not cognitive impairment or cortical tissue loss following traumatic brain injury in ratsJ A Cheney, A L Brown, F M Bareyre, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|April 27, 2001
The maxi-K channel opener BMS-204352 attenuates regional cerebral edema and neurologic motor impairment after experimental brain injuryJ A Cheney, J D Weisser, F M Bareyre, et al.
Journal of Neurotrauma|April 1, 1997
Delayed administration of basic fibroblast growth factor (bFGF) attenuates cognitive dysfunction following parasagittal fluid percussion brain injury in the ratK L McDermott, R Raghupathi, S C Fernandez, et al.
Experimental Neurology|May 1, 1999
Genetic approaches to neurotrauma research: opportunities and potential pitfalls of murine modelsO Steward, P E Schauwecker, L Guth, et al.
Experimental Neurology|April 29, 2000
Brain trauma in aged transgenic mice induces regression of established abeta depositsY Nakagawa, L Reed, M Nakamura, et al.
Journal of Neurosurgery|November 13, 2001
Mild head injury increasing the brain's vulnerability to a second concussive impactH L Laurer, F M Bareyre, V M Lee, et al.
The Journal of Comparative Neurology|March 26, 1998
Twofold overexpression of human beta-amyloid precursor proteins in transgenic mice does not affect the neuromotor, cognitive, or neurodegenerative sequelae following experimental brain injuryH Murai, J E Pierce, R Raghupathi, et al.
The American Journal of Pathology|September 15, 1998
Brain trauma induces massive hippocampal neuron death linked to a surge in beta-amyloid levels in mice overexpressing mutant amyloid precursor proteinD H Smith, M Nakamura, T K McIntosh, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|November 10, 2001
A review and rationale for the use of genetically engineered animals in the study of traumatic brain injuryL Longhi, K E Saatman, R Raghupathi, et al.
The Journal of Comparative Neurology|July 22, 1999
Traumatic brain injury in young, amyloid-beta peptide overexpressing transgenic mice induces marked ipsilateral hippocampal atrophy and diminished Abeta deposition during agingY Nakagawa, M Nakamura, T K McIntosh, et al.
Pageof 8