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Cancer Research|February 15, 1997
Preclinical studies with Fc(gamma)R bispecific antibodies and granulocyte colony-stimulating factor-primed neutrophils as effector cells against HER-2/neu overexpressing breast cancerB Stockmeyer, T Valerius, R Repp, et al.Cytometry. Part B, Clinical Cytometry|April 30, 2003
Immunophenotyping is an independent factor for risk stratification in AMLR Repp, U Schaekel, G Helm, et al.Annals of the Rheumatic Diseases|August 15, 2002
Fcgamma receptor IIa, IIIa, and IIIb polymorphisms in German patients with systemic lupus erythematosus: association with clinical symptomsK Manger, R Repp, M Jansen, et al.Blood|May 1, 1996
HLA class II as potential target antigen on malignant B cells for therapy with bispecific antibodies in combination with granulocyte colony-stimulating factorD Elsässer, T Valerius, R Repp, et al.Arthritis and Rheumatism|July 15, 1998
Fcgamma receptor IIa polymorphism in Caucasian patients with systemic lupus erythematosus: association with clinical symptomsK Manger, R Repp, B M Spriewald, et al.Oncogene|September 1, 1999
A DNA damage repair mechanism is involved in the origin of chromosomal translocations t(4;11) in primary leukemic cellsE Gillert, T Leis, R Repp, et al.Cancer Research|July 29, 1998
Evaluating antibodies for their capacity to induce cell-mediated lysis of malignant B cellsD Würflein, M Dechant, B Stockmeyer, et al.Annals of Oncology : Official Journal of the European Society for Medical Oncology|November 3, 2004
Rituximab consolidation after high-dose chemotherapy and autologous blood stem cell transplantation in follicular and mantle cell lymphoma: a prospective, multicenter phase II studyW Brugger, J Hirsch, F Grünebach, et al.Journal of Immunology (Baltimore, Md. : 1950)|November 9, 2000
Triggering Fc alpha-receptor I (CD89) recruits neutrophils as effector cells for CD20-directed antibody therapyB Stockmeyer, M Dechant, M van Egmond, et al.Proceedings of the National Academy of Sciences of the United States of America|July 26, 2000
The human GRAF gene is fused to MLL in a unique t(5;11)(q31;q23) and both alleles are disrupted in three cases of myelodysplastic syndrome/acute myeloid leukemia with a deletion 5qA Borkhardt, S Bojesen, O A Haas, et al.Pageof 8