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Toxicological Sciences : an Official Journal of the Society of Toxicology|May 14, 1998
Tissue repair response as a function of dose during trichloroethylene hepatotoxicityM G Soni, R S Mangipudy, M M Mumtaz, et al.Medical Hypotheses|March 1, 1996
Nutritional modulation of the final outcome of hepatotoxic injury by energy substrates: an hypothesis for the mechanismS Chanda, H M Mehendale, S ChandraMolecular and Cellular Biochemistry|November 24, 1999
Role of nitric oxide in the induction of apoptosis by smokeless tobacco extractR S Mangipudy, J K VishwanathaFundamental and Applied Toxicology : Official Journal of the Society of Toxicology|June 1, 1984
Potentiation of halomethane hepatotoxicity: chlordecone and carbon tetrachlorideH M MehendaleEnvironmental Health Perspectives|December 1, 1977
Chemical reactivity-absorption, retention, metabolism, and elimination of hexachlorocyclopentadieneH M MehendaleDrug Metabolism and Disposition: the Biological Fate of Chemicals|March 1, 1976
Uptake and disposition of chlorinated biphenyls by isolated perfused rat liverH M MehendaleToxicology Letters|December 1, 1989
Mechanism of the lethal interaction of chlordecone and CCl4 at non-toxic dosesH M MehendaleToxicological Sciences : an Official Journal of the Society of Toxicology|September 28, 2000
PPAR-alpha: a key to the mechanism of hepatoprotection by clofibrateH M MehendaleToxicology|December 28, 1995
Toxicodynamics of low level toxicant interactions of biological significance: inhibition of tissue repairH M MehendaleDrug Metabolism and Disposition: the Biological Fate of Chemicals|January 1, 1977
Mirex-induced impairment of hepatobiliary function. Suppressed biliary excretion of imipramine and sulfobromophthaleinH M MehendalePageof 21