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Journal of Neural Transmission (Vienna, Austria : 1996)|March 3, 2004
Differential effects of "Advanced glycation endproducts" and beta-amyloid peptide on glucose utilization and ATP levels in the neuronal cell line SH-SY5YB Kuhla, C Loske, S Garcia De Arriba, et al.
Journal of Neural Transmission. Parkinson'S Disease and Dementia Section|January 1, 1994
The cognition-enhancing drug tenilsetam is an inhibitor of protein crosslinking by advanced glycosylationG Münch, Y Taneli, E Schraven, et al.
Kidney International. Supplement|February 13, 2001
Circulating advanced glycation end product levels in rats rapidly increase with acute renal failureK Sebeková, P Blazícek, D Syrová, et al.
American Journal of Kidney Diseases : the Official Journal of the National Kidney Foundation|September 29, 2001
N(epsilon)-(carboxymethyl)lysine levels in patients with type 2 diabetes: role of renal functionZ Wagner, I Wittmann, I Mazák, et al.
Journal of Neural Transmission (Vienna, Austria : 1996)|December 30, 1998
Cytotoxicity of advanced glycation endproducts is mediated by oxidative stressC Loske, A Neumann, A M Cunningham, et al.
Clinical Nephrology|November 15, 2000
Low-molecular but not high-molecular advanced glycation end products (AGEs) are removed by high-flux dialysisA Gerdemann, H D Lemke, A Nothdurft, et al.
European Journal of Biochemistry|June 24, 2000
Transition metal-mediated glycoxidation accelerates cross-linking of beta-amyloid peptideC Loske, A Gerdemann, W Schepl, et al.
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