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The Journal of Investigative Dermatology|June 10, 1998
Ex vivo and in vivo adenovirus-mediated gene therapy strategies induce a systemic anti-tumor immune defence in the B16 melanoma modelB Bonnekoh, D A Greenhalgh, S H Chen, et al.
Cancer Research|November 1, 1993
Cooperation between v-fos and v-rasHA induces autonomous papillomas in transgenic epidermis but not malignant conversionD A Greenhalgh, M I Quintanilla, C C Orengo, et al.
Differentiation; Research in Biological Diversity|December 1, 1996
A transgenic mouse model that recapitulates the clinical features of both neonatal and adult forms of the skin disease epidermolytic hyperkeratosisJ R Bickenbach, M A Longley, D S Bundman, et al.
Prenatal Diagnosis|September 22, 1998
Prenatal diagnosis for keratin mutations to exclude transmission of epidermolytic hyperkeratosisJ A Rothnagel, M T Lin, M A Longley, et al.
The Journal of Investigative Dermatology|April 14, 1999
A novel substitution in keratin 10 in epidermolytic hyperkeratosisM J Arin, M A Longley, I Anton-Lamprecht, et al.
Nature Genetics|August 1, 1994
Mutations in the rod domain of keratin 2e in patients with ichthyosis bullosa of SiemensJ A Rothnagel, H Traupe, S Wojcik, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 18, 1997
Expression of a dominant-negative type II transforming growth factor beta (TGF-beta) receptor in the epidermis of transgenic mice blocks TGF-beta-mediated growth inhibitionX J Wang, D A Greenhalgh, J R Bickenbach, et al.
Cancer Research|May 1, 1995
Autocrine transforming growth factor alpha is dispensible for v-rasHa-induced epidermal neoplasia: potential involvement of alternate epidermal growth factor receptor ligandsA A Dlugosz, C Cheng, E K Williams, et al.
Immunogenetics|January 1, 1987
Assignment of the Ly-6--Ril-1--Sis--H-30--Pol-5/Xmmv-72--Ins-3--Krt-1--Int-1 --Gdc-1 region to mouse chromosome 15D Meruelo, A Rossomando, S Scandalis, et al.
The Journal of Cell Biology|October 19, 2000
Transgenic mice expressing a mutant form of loricrin reveal the molecular basis of the skin diseases, Vohwinkel syndrome and progressive symmetric erythrokeratodermaY Suga, M Jarnik, P S Attar, et al.
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