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The Journal of Experimental Medicine|August 1, 1983
Generation of three different fragments of bound C3 with purified factor I or serum. II. Location of binding sites in the C3 fragments for factors B and H, complement receptors, and bovine conglutininG D Ross, S L Newman, J D Lambris, et al.Annals of Human Genetics|April 1, 1995
Complement component C6 and C7 haplotypes associated with deficiencies of C6B A Fernie, A Orren, R Würzner, et al.Journal of Neuroimmunology|June 22, 1999
Comparison of C1q-receptors on rat microglia and peritoneal macrophagesM G Wing, D J Seilly, R S Nicholas, et al.Clinical and Experimental Immunology|June 1, 1997
Difficulties in the ascertainment of C9 deficiency: lessons to be drawn from a compound heterozygote C9-deficient subjectM J Hobart, B A Fernie, R Würzner, et al.Clinical and Experimental Immunology|February 1, 1995
The immunosuppressive drug thalidomide induces T helper cell type 2 (Th2) and concomitantly inhibits Th1 cytokine production in mitogen- and antigen-stimulated human peripheral blood mononuclear cell culturesS M McHugh, I R Rifkin, J Deighton, et al.Clinical and Experimental Immunology|February 1, 1991
The complement-inhibiting protein, protectin (CD59 antigen), is present and functionally active on glomerular epithelial cellsI A Rooney, A Davies, D Griffiths, et al.The Journal of Biological Chemistry|August 15, 1989
A library of oligosaccharide probes (neoglycolipids) from N-glycosylated proteins reveals that conglutinin binds to certain complex-type as well as high mannose-type oligosaccharide chainsT Mizuochi, R W Loveless, A M Lawson, et al.The Journal of Clinical Investigation|March 1, 1987
Leukocytes from four patients with complete or partial Leu-CAM deficiency contain the common beta-subunit precursor and beta-subunit messenger RNAN Dana, L K Clayton, D G Tennen, et al.Clinical and Experimental Immunology|July 9, 2020
Primary pneumococcal peritonitis can be the first presentation of a familial complement factor I deficiency1S Ugrinovic, H Firth, D Kavanagh, et al.The Journal of Clinical Investigation|December 15, 1996
Mechanism of first-dose cytokine-release syndrome by CAMPATH 1-H: involvement of CD16 (FcgammaRIII) and CD11a/CD18 (LFA-1) on NK cellsM G Wing, T Moreau, J Greenwood, et al.Pageof 23