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Angiogenesis|June 8, 2020
Arterial endoglin does not protect against arteriovenous malformationsEsha Singh, Rachael E Redgrave, Helen M Phillips, et al.
Frontiers in Aging|December 1, 2022
Anthracycline-induced cardiotoxicity and senescenceLaura K Booth, Rachael E Redgrave, Omowumi Folaranmi, et al.
Cells|February 23, 2024
Cellular Senescence, Mitochondrial Dysfunction, and Their Link to Cardiovascular DiseaseMaria Camacho-Encina, Laura K Booth, Rachael E Redgrave, et al.
Sub-Cellular Biochemistry|April 29, 2023
Heart Disease and Ageing: The Roles of Senescence, Mitochondria, and Telomerase in Cardiovascular DiseaseLaura K Booth, Rachael E Redgrave, Simon Tual-Chalot, et al.
Plos One|June 5, 2014
Endothelial depletion of Acvrl1 in mice leads to arteriovenous malformations associated with reduced endoglin expressionSimon Tual-Chalot, Marwa Mahmoud, Kathleen R Allinson, et al.
Stem Cell Reports|May 13, 2017
Cardiosphere-Derived Cells Require Endoglin for Paracrine-Mediated AngiogenesisRachael E Redgrave, Simon Tual-Chalot, Benjamin J Davison, et al.
International Journal of Cardiology. Heart & Vasculature|November 25, 2016
Using MRI to predict future adverse cardiac remodelling in a male mouse model of myocardial infarctionRachael E Redgrave, Simon Tual-Chalot, Benjamin J Davison, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|April 29, 2021
Rapid fall in circulating non-classical monocytes in ST elevation myocardial infarction patients correlates with cardiac injurySarah A Marsh, Catherine Park, Rachael E Redgrave, et al.
Npj Aging|June 14, 2023
Senescent cardiomyocytes contribute to cardiac dysfunction following myocardial infarctionRachael E Redgrave, Emily Dookun, Laura K Booth, et al.
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