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Journal of Psychiatric Research|February 17, 2019
Clinical and genetic analysis of children with a dual diagnosis of Tourette syndrome and autism spectrum disorderKarin Vanessa Carias, Rachel WevrickMolecular Therapy. Methods & Clinical Development|April 17, 2019
Preclinical Testing in Translational Animal Models of Prader-Willi Syndrome: Overview and Gap AnalysisK Vanessa Carias, Rachel WevrickMolecular Genetics and Genomics : MGG|January 13, 2018
Genetic analysis of very obese children with autism spectrum disorderHerman D Cortes, Rachel WevrickGene|February 7, 2012
Loss of the Prader-Willi obesity syndrome protein necdin promotes adipogenesisJason Russell Bush, Rachel WevrickEndocrinology|January 21, 2011
Impaired hypothalamic regulation of endocrine function and delayed counterregulatory response to hypoglycemia in Magel2-null miceAlysa A Tennese, Rachel WevrickPlos One|January 28, 2009
Loss of magel2, a candidate gene for features of Prader-Willi syndrome, impairs reproductive function in miceRebecca E Mercer, Rachel WevrickGenesis (New York, N.Y. : 2000)|July 29, 2010
Loss of Necdin impairs myosin activation and delays cell polarizationJason R Bush, Rachel WevrickMolecular Genetics and Metabolism|March 7, 2018
Chronic diazoxide treatment decreases fat mass and improves endurance capacity in an obese mouse model of Prader-Willi syndromeJocelyn M Bischof, Rachel WevrickPhysiological Genomics|May 5, 2005
Genome-wide analysis of gene transcription in the hypothalamusJocelyn M Bischof, Rachel WevrickPageof 5