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Plos One|April 22, 2020
A MAGEL2-deubiquitinase complex modulates the ubiquitination of circadian rhythm protein CRY1K Vanessa Carias, Mercedes Zoeteman, Abigail Seewald, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 12, 2003
Absence of Ndn, encoding the Prader-Willi syndrome-deleted gene necdin, results in congenital deficiency of central respiratory drive in neonatal miceJun Ren, Syann Lee, Silvia Pagliardini, et al.
American Journal of Physiology. Cell Physiology|October 19, 2012
Claudin-4 forms a paracellular barrier, revealing the interdependence of claudin expression in the loose epithelial cell culture model opossum kidney cellsJelena Borovac, Reid S Barker, Juraj Rievaj, et al.
American Journal of Medical Genetics. Part B, Neuropsychiatric Genetics : the Official Publication of the International Society of Psychiatric Genetics|February 10, 2009
Regionally reduced brain volume, altered serotonin neurochemistry, and abnormal behavior in mice null for the circadian rhythm output gene Magel2Rebecca E Mercer, Erin M Kwolek, Jocelyn M Bischof, et al.
Human Molecular Genetics|October 4, 2017
The Prader-Willi syndrome proteins MAGEL2 and necdin regulate leptin receptor cell surface abundance through ubiquitination pathwaysTishani Methsala Wijesuriya, Leentje De Ceuninck, Delphine Masschaele, et al.
Plos Genetics|January 24, 2013
Magel2 is required for leptin-mediated depolarization of POMC neurons in the hypothalamic arcuate nucleus in miceRebecca E Mercer, Sheldon D Michaelson, Melissa J S Chee, et al.
Human Molecular Genetics|January 15, 2005
Essential role for the Prader-Willi syndrome protein necdin in axonal outgrowthSyann Lee, Christine L Walker, Barbara Karten, et al.
Molecular Therapy. Methods & Clinical Development|October 26, 2022
Hypothalamic AAV-BDNF gene therapy improves metabolic function and behavior in the <i>Magel2</i>-null mouse model of Prader-Willi syndromeNicholas J Queen, Xunchang Zou, Jacqueline M Anderson, et al.
Plos One|April 5, 2013
The Smc5/Smc6/MAGE complex confers resistance to caffeine and genotoxic stress in Drosophila melanogasterXiao Li, Ran Zhuo, Stanley Tiong, et al.
Human Molecular Genetics|July 21, 2016
Muscle dysfunction caused by loss of Magel2 in a mouse model of Prader-Willi and Schaaf-Yang syndromesAin A Kamaludin, Christa Smolarchuk, Jocelyn M Bischof, et al.
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