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Orphanet Journal of Rare Diseases|July 22, 2018
ROHHAD and Prader-Willi syndrome (PWS): clinical and genetic comparisonSarah F Barclay, Casey M Rand, Lisa Nguyen, et al.Blood|July 11, 2012
Necdin, a p53 target gene, regulates the quiescence and response to genotoxic stress of hematopoietic stem/progenitor cellsTakashi Asai, Yan Liu, Silvana Di Giandomenico, et al.Nature Genetics|September 26, 2007
The imprinted gene Magel2 regulates normal circadian outputSerguei V Kozlov, James W Bogenpohl, Maureen P Howell, et al.Plos Genetics|September 17, 2013
Stochastic loss of silencing of the imprinted Ndn/NDN allele, in a mouse model and humans with prader-willi syndrome, has functional consequencesAnne Rieusset, Fabienne Schaller, Unga Unmehopa, et al.Molecular Metabolism|December 1, 2016
Targeting the endocannabinoid/CB1 receptor system for treating obesity in Prader-Willi syndromeIbrahim Knani, Brian J Earley, Shiran Udi, et al.Nature Neuroscience|July 15, 2020
Regulation of autism-relevant behaviors by cerebellar-prefrontal cortical circuitsElyza Kelly, Fantao Meng, Hirofumi Fujita, et al.Obesity (Silver Spring, Md.)|February 28, 2014
Hyperphagia: current concepts and future directions proceedings of the 2nd international conference on hyperphagiaSteven B Heymsfield, Nicole M Avena, Leslie Baier, et al.Biorxiv : the Preprint Server for Biology|June 26, 2025
Assigning Targetable Molecular Pathways to Transdiagnostic Subgroups Across Autism and Related Neurodevelopmental DisordersJacob Ellegood, Antoine Beauchamp, Yohan Yee, et al.Pageof 5