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The Journal of Physiology|August 5, 2003
Protein phosphatases decrease sarcoplasmic reticulum calcium content by stimulating calcium release in cardiac myocytesDmitry Terentyev, Serge Viatchenko-Karpinski, Inna Gyorke, et al.
Journal of Molecular and Cellular Cardiology|April 15, 2021
Sarcoplasmic reticulum-mitochondria communication; implications for cardiac arrhythmiaShanna Hamilton, Radmila Terentyeva, Richard T Clements, et al.
American Journal of Physiology. Heart and Circulatory Physiology|January 2, 2014
Sarcoplasmic reticulum Ca²⁺ release is both necessary and sufficient for SK channel activation in ventricular myocytesDmitry Terentyev, Jennifer A Rochira, Radmila Terentyeva, et al.
The Journal of Physiology|September 18, 2013
Redox modification of ryanodine receptors by mitochondria-derived reactive oxygen species contributes to aberrant Ca2+ handling in ageing rabbit heartsLeroy L Cooper, Weiyan Li, Yichun Lu, et al.
The Journal of Physiology|August 3, 2011
Arrhythmogenic adverse effects of cardiac glycosides are mediated by redox modification of ryanodine receptorsHsiang-Ting Ho, Sarah C W Stevens, Radmila Terentyeva, et al.
The Journal of Physiology|October 16, 2004
Modulation of cytosolic and intra-sarcoplasmic reticulum calcium waves by calsequestrin in rat cardiac myocytesZuzana Kubalova, Inna Györke, Radmila Terentyeva, et al.
Cardiovascular Research|January 29, 2011
The relationship between arrhythmogenesis and impaired contractility in heart failure: role of altered ryanodine receptor functionAndriy E Belevych, Dmitry Terentyev, Radmila Terentyeva, et al.
Circulation Research|February 26, 2005
Triadin overexpression stimulates excitation-contraction coupling and increases predisposition to cellular arrhythmia in cardiac myocytesDmitry Terentyev, Steven E Cala, Timothy D Houle, et al.
Biophysical Journal|July 16, 2020
Impact of I<sub>SK</sub> Voltage and Ca<sup>2+</sup>/Mg<sup>2+</sup>-Dependent Rectification on Cardiac RepolarizationPeter Bronk, Tae Yun Kim, Iuliia Polina, et al.
Basic Research in Cardiology|June 15, 2017
The role of spatial organization of Ca<sup>2+</sup> release sites in the generation of arrhythmogenic diastolic Ca<sup>2+</sup> release in myocytes from failing heartsAndriy E Belevych, Hsiang-Ting Ho, Ingrid M Bonilla, et al.
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