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Antioxidants & Redox Signaling|October 4, 2008
Connexin-caused genetic diseases and corresponding mouse modelsRadoslaw Dobrowolski, Klaus Willecke
The Journal of Membrane Biology|August 10, 2007
Some oculodentodigital dysplasia-associated Cx43 mutations cause increased hemichannel activity in addition to deficient gap junction channelsRadoslaw Dobrowolski, Annette Sommershof, Klaus Willecke
Developmental Dynamics : an Official Publication of the American Association of Anatomists|June 24, 2008
Hepatoma-derived growth factor (HDGF) is dispensable for normal mouse developmentRainer Gallitzendoerfer, Mekky M Abouzied, Dieter Hartmann, et al.
Science Signaling|January 26, 2012
Extracellular Ca²⁺ acts as a mediator of communication from neurons to gliaArnulfo Torres, Fushun Wang, Qiwu Xu, et al.
Human Molecular Genetics|April 21, 2007
The connexin31 F137L mutant mouse as a model for the human skin disease erythrokeratodermia variabilis (EKV)Marc Schnichels, Philipp Wörsdörfer, Radoslaw Dobrowolski, et al.
Brain Research Bulletin|July 10, 2017
Connexin43, but not connexin30, contributes to adult neurogenesis in the dentate gyrusJiong Zhang, Stephanie Griemsmann, Zhou Wu, et al.
European Journal of Cell Biology|October 14, 2008
Mouse lens connexin23 (Gje1) does not form functional gap junction channels but causes enhanced ATP release from HeLa cellsStephan Sonntag, Goran Söhl, Radoslaw Dobrowolski, et al.
Experimental Cell Research|February 13, 2009
The TSG101 protein binds to connexins and is involved in connexin degradationTanja Auth, Sharazad Schlüter, Stephanie Urschel, et al.
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