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Human Molecular Genetics|January 9, 2022
Abnormal activation of Yap/Taz contributes to the pathogenesis of tuberous sclerosis complexBethany K Terry, Raehee Park, Seo-Hee Cho, et al.Nature Communications|January 5, 2023
An entosis-like process induces mitotic disruption in Pals1 microcephaly pathogenesisNoelle A Sterling, Jun Young Park, Raehee Park, et al.Developmental Biology|September 13, 2016
Yap is essential for retinal progenitor cell cycle progression and RPE cell fate acquisition in the developing mouse eyeJin Young Kim, Raehee Park, Jin Hwan J Lee, et al.Nature Communications|January 13, 2016
Yap is required for ependymal integrity and is suppressed in LPA-induced hydrocephalusRaehee Park, Uk Yeol Moon, Jun Young Park, et al.Developmental Biology|January 4, 2014
Dual function of Yap in the regulation of lens progenitor cells and cellular polarityJi Yun Song, Raehee Park, Jin Young Kim, et al.Developmental Biology|October 7, 2019
Yap/Taz are required for establishing the cerebellar radial glia scaffold and proper foliationLucinda J Hughes, Raehee Park, Min Jung Lee, et al.Human Molecular Genetics|January 23, 2019
Nearly complete deletion of BubR1 causes microcephaly through shortened mitosis and massive cell deathAmbrosia J Simmons, Raehee Park, Noelle A Sterling, et al.The Journal of Nutritional Biochemistry|February 5, 2011
Green tea (-)-epigallocatechin-3-gallate inhibits HGF-induced progression in oral cavity cancer through suppression of HGF/c-MetYoon Woo Koh, Eun Chang Choi, Sung Un Kang, et al.Development (Cambridge, England)|December 15, 2015
The apical complex protein Pals1 is required to maintain cerebellar progenitor cells in a proliferative stateJun Young Park, Lucinda J Hughes, Uk Yeol Moon, et al.Cell Reports|August 4, 2015
Impaired Reelin-Dab1 Signaling Contributes to Neuronal Migration Deficits of Tuberous Sclerosis ComplexUk Yeol Moon, Jun Young Park, Raehee Park, et al.Pageof 2