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Plos One|March 1, 2019
The HERV-K accessory protein Np9 controls viability and migration of teratocarcinoma cellsSusana M Chan, Tamar Sapir, Sung-Soo Park, et al.BMC Genomics|December 15, 2010
NGSQC: cross-platform quality analysis pipeline for deep sequencing dataManhong Dai, Robert C Thompson, Christopher Maher, et al.Scientific Reports|August 4, 2019
The Genomic Landscape of Centromeres in CancersAnjan K Saha, Mohamad Mourad, Mark H Kaplan, et al.Biorxiv : the Preprint Server for Biology|October 24, 2023
Fyn-mediated phosphorylation of Menin disrupts telomere maintenance in stem cellsSouren Paul, Preston M McCourt, Le Thi My Le, et al.Journal of Virology|June 13, 2014
Genomic flexibility of human endogenous retrovirus type KDerek Dube, Rafael Contreras-Galindo, Shirley He, et al.Journal of Virology|May 18, 2012
Expression of human endogenous retrovirus type K (HML-2) is activated by the Tat protein of HIV-1Marta J Gonzalez-Hernandez, Michael D Swanson, Rafael Contreras-Galindo, et al.Journal of Virology|May 1, 2015
Human Endogenous Retrovirus Type K (HERV-K) Particles Package and Transmit HERV-K-Related SequencesRafael Contreras-Galindo, Mark H Kaplan, Derek Dube, et al.Journal of Virology|July 18, 2008
Human endogenous retrovirus K (HML-2) elements in the plasma of people with lymphoma and breast cancerRafael Contreras-Galindo, Mark H Kaplan, Philippe Leissner, et al.Infectious Agents and Cancer|March 14, 2020
Is the HERV-K HML-2 Xq21.33, an endogenous retrovirus mutated by gene conversion of chromosome X in a subset of African populations, associated with human breast cancer?Mark H Kaplan, Rafael Contreras-Galindo, Evelyn Jiagge, et al.Genome Biology|April 18, 2015
Expansion of a novel endogenous retrovirus throughout the pericentromeres of modern humansJoseph Zahn, Mark H Kaplan, Sabrina Fischer, et al.Pageof 4