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The Journal of Biological Chemistry|February 16, 2012
EMILIN-3, peculiar member of elastin microfibril interface-located protein (EMILIN) family, has distinct expression pattern, forms oligomeric assemblies, and serves as transforming growth factor β (TGF-β) antagonistAlvise Schiavinato, Ann-Kathrin A Becker, Miriam Zanetti, et al.
The Journal of Biological Chemistry|November 18, 2017
The cartilage-specific lectin C-type lectin domain family 3 member A (CLEC3A) enhances tissue plasminogen activator-mediated plasminogen activationDaniela Lau, Dzemal Elezagic, Gabriele Hermes, et al.
The Journal of Biological Chemistry|February 25, 2014
A mouse model for dominant collagen VI disorders: heterozygous deletion of Col6a3 Exon 16Te-Cheng Pan, Rui-Zhu Zhang, Machiko Arita, et al.
The Journal of Investigative Dermatology|October 1, 2010
Expression of the collagen VI α5 and α6 chains in normal human skin and in skin of patients with collagen VI-related myopathiesPatrizia Sabatelli, Sudheer K Gara, Paolo Grumati, et al.
The Journal of Clinical Investigation|October 21, 2014
Axonally derived matrilin-2 induces proinflammatory responses that exacerbate autoimmune neuroinflammationAnna Jonas, Stefan Thiem, Tanja Kuhlmann, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|April 22, 2022
LTBP1 promotes fibrillin incorporation into the extracellular matrixMatthias Przyklenk, Veronika S Georgieva, Fabian Metzen, et al.
International Journal of Molecular Sciences|January 23, 2020
Mice Lacking the Matrilin Family of Extracellular Matrix Proteins Develop Mild Skeletal Abnormalities and Are Susceptible to Age-Associated OsteoarthritisPing Li, Lutz Fleischhauer, Claudia Nicolae, et al.
Human Mutation|April 14, 2025
The UCMD-Causing <i>COL6A1</i> (<i>c</i>.930 + 189<i>C</i> > <i>T</i>) Intron Mutation Leads to the Secretion and Aggregation of Single Mutated Collagen VI <i>α</i>1 ChainsCarolin D Freiburg, Herimela Solomon-Degefa, Patrick Freiburg, et al.
Human Molecular Genetics|May 23, 2007
Decreased chondrocyte proliferation and dysregulated apoptosis in the cartilage growth plate are key features of a murine model of epiphyseal dysplasia caused by a matn3 mutationMatthew P Leighton, Seema Nundlall, Tobias Starborg, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|January 10, 2012
Expression of collagen VI α5 and α6 chains in human muscle and in Duchenne muscular dystrophy-related muscle fibrosisPatrizia Sabatelli, Francesca Gualandi, Sudheer Kumar Gara, et al.
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