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Nature Protocols|November 10, 2019
Decellularization and antibody staining of mouse tissues to map native extracellular matrix structures in 3DAlejandro E Mayorca-Guiliani, Oliver Willacy, Chris D Madsen, et al.
The Journal of Investigative Dermatology|November 16, 2013
AMACO is a component of the basement membrane-associated Fraser complexRebecca J Richardson, Jan M Gebauer, Jin-Li Zhang, et al.
Nature Protocols|March 24, 2020
Author Correction: Decellularization and antibody staining of mouse tissues to map native extracellular matrix structures in 3DAlejandro E Mayorca-Guiliani, Oliver Willacy, Chris D Madsen, et al.
The Journal of Biological Chemistry|July 29, 2020
Structure of a collagen VI α3 chain VWA domain array: adaptability and functional implications of myopathy causing mutationsHerimela Solomon-Degefa, Jan M Gebauer, Cy M Jeffries, et al.
Plos Genetics|April 27, 2010
Genetic analysis of fin development in zebrafish identifies furin and hemicentin1 as potential novel fraser syndrome disease genesThomas J Carney, Natália Martins Feitosa, Carmen Sonntag, et al.
Human Molecular Genetics|May 6, 2017
The chaperone activity of 4PBA ameliorates the skeletal phenotype of Chihuahua, a zebrafish model for dominant osteogenesis imperfectaRoberta Gioia, Francesca Tonelli, Ilaria Ceppi, et al.
Immunity|October 18, 2015
Interleukin-4 Receptor α Signaling in Myeloid Cells Controls Collagen Fibril Assembly in Skin RepairJohanna A Knipper, Sebastian Willenborg, Jürgen Brinckmann, et al.
Matrix Biology : Journal of the International Society for Matrix Biology|November 17, 2025
Furin-like cleavage at the C1-C2 linker region of the ⍺3 chain is not required for collagen VI assemblyArthur Lauri Pasanen-Zentz, Mengjie Zhu, Sebastian Schmitz, et al.
Plos One|January 20, 2018
A homozygous missense variant in VWA2, encoding an interactor of the Fraser-complex, in a patient with vesicoureteral refluxAmelie T van der Ven, Birgit Kobbe, Stefan Kohl, et al.
JCI Insight|March 22, 2019
A recurrent COL6A1 pseudoexon insertion causes muscular dystrophy and is effectively targeted by splice-correction therapiesVéronique Bolduc, A Reghan Foley, Herimela Solomon-Degefa, et al.
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