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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|January 10, 2020
FIH-1 engages novel binding partners to positively influence epithelial proliferation via p63Nihal Kaplan, Ying Dong, Sijia Wang, et al.Proceedings of the National Academy of Sciences of the United States of America|March 23, 2021
Mitochondrial metabolism is essential for invariant natural killer T cell development and functionXiufang Weng, Amrendra Kumar, Liang Cao, et al.American Journal of Physiology. Lung Cellular and Molecular Physiology|September 9, 2005
Bleomycin induces alveolar epithelial cell death through JNK-dependent activation of the mitochondrial death pathwayVivian Y Lee, Clara Schroedl, Joslyn K Brunelle, et al.Molecular and Cellular Biology|December 6, 2006
Loss of Mcl-1 protein and inhibition of electron transport chain together induce anoxic cell deathJoslyn K Brunelle, Emelyn H Shroff, Harris Perlman, et al.Proceedings of the National Academy of Sciences of the United States of America|January 4, 2008
A chemical genomics screen highlights the essential role of mitochondria in HIF-1 regulationXiaoyu Lin, Caroline A David, Jennifer B Donnelly, et al.Nature Communications|August 5, 2024
Hif1α-dependent mitochondrial acute O2 sensing and signaling to myocyte Ca2+ channels mediate arterial hypoxic vasodilationAlejandro Moreno-Domínguez, Olalla Colinas, Ignacio Arias-Mayenco, et al.Circulation Research|April 15, 2006
Hypoxia-mediated degradation of Na,K-ATPase via mitochondrial reactive oxygen species and the ubiquitin-conjugating systemAlejandro P Comellas, Laura A Dada, Emilia Lecuona, et al.Immunity|February 4, 2023
Oxidative phosphorylation selectively orchestrates tissue macrophage homeostasisStefanie K Wculek, Ignacio Heras-Murillo, Annalaura Mastrangelo, et al.Cell Reports|July 4, 2019
Requirement of Mitochondrial Transcription Factor A in Tissue-Resident Regulatory T Cell Maintenance and FunctionZheng Fu, Jian Ye, Joseph W Dean, et al.FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|October 25, 2008
Hyperoxia-induced premature senescence requires p53 and pRb, but not mitochondrial matrix ROSTatyana A Klimova, Eric L Bell, Emelyn H Shroff, et al.Pageof 29