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Hematology/Oncology Clinics of North America|November 13, 2007
Immune therapiesRao H Prabhala, Nikhil C Munshi
Plos One|May 29, 2014
CCR6, the sole receptor for the chemokine CCL20, promotes spontaneous intestinal tumorigenesisBisweswar Nandi, Christine Pai, Qin Huang, et al.
Oncotarget|December 2, 2021
CCL20 induces colorectal cancer neoplastic epithelial cell proliferation, migration, and further CCL20 production through autocrine HGF-c-Met and MSP-MSPR signaling pathwaysBisweswar Nandi, Jonathan Pastrana Del Valle, Mehmet K Samur, et al.
Cancer Immunology, Immunotherapy : CII|November 13, 2015
Deficiency of IL-17A, but not the prototypical Th17 transcription factor RORγt, decreases murine spontaneous intestinal tumorigenesisMia Shapiro, Bisweswar Nandi, Christine Pai, et al.
Oncoimmunology|February 16, 2017
Deficiency of the immunostimulatory cytokine IL-21 promotes intestinal neoplasia via dysregulation of the Th1/Th17 axisMia Shapiro, Bisweswar Nandi, Gabriel Gonzalez, et al.
Blood Cancer Journal|October 9, 2021
Dysregulated APOBEC3G causes DNA damage and promotes genomic instability in multiple myelomaSrikanth Talluri, Mehmet K Samur, Leutz Buon, et al.
Oncoimmunology|September 14, 2016
Stromal CCR6 drives tumor growth in a murine transplantable colon cancer through recruitment of tumor-promoting macrophagesBisweswar Nandi, Mia Shapiro, Mehmet K Samur, et al.
Scientific Reports|April 5, 2017
Isolation of Circulating Plasma Cells in Multiple Myeloma Using CD138 Antibody-Based Capture in a Microfluidic DeviceMohammad A Qasaimeh, Yichao C Wu, Suman Bose, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 19, 2008
MHC class I chain-related protein A antibodies and shedding are associated with the progression of multiple myelomaMasahisa Jinushi, Matthew Vanneman, Nikhil C Munshi, et al.
Blood|December 8, 2009
The sumoylation pathway is dysregulated in multiple myeloma and is associated with adverse patient outcomeJames J Driscoll, Dheeraj Pelluru, Konstantinos Lefkimmiatis, et al.
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