Showing results (1-10 of 300) with videos related to
Sort By:
Pageof 30
International Journal of Molecular Sciences|August 3, 2017
Do Multiwell Plate High Throughput Assays Measure Loss of Cell Viability Following Exposure to Genotoxic Agents?Razmik Mirzayans, Bonnie Andrais, David MurrayCancers|April 18, 2018
Roles of Polyploid/Multinucleated Giant Cancer Cells in Metastasis and Disease Relapse Following Anticancer TreatmentRazmik Mirzayans, Bonnie Andrais, David MurrayInternational Journal of Molecular Sciences|July 8, 2017
Impact of Premature Senescence on Radiosensitivity Measured by High Throughput Cell-Based AssaysRazmik Mirzayans, Bonnie Andrais, David MurrayMethods in Molecular Biology (Clifton, N.J.)|June 24, 2024
Single-Cell MTT: A Simple and Sensitive Assay for Determining the Viability and Metabolic Activity of Polyploid Giant Cancer Cells (PGCCs)Razmik Mirzayans, Bonnie Andrais, David MurrayCancers|August 4, 2018
Viability Assessment Following Anticancer Treatment Requires Single-Cell VisualizationRazmik Mirzayans, Bonnie Andrais, David MurrayBiochemistry Research International|August 28, 2012
Role of p16(INK4A) in Replicative Senescence and DNA Damage-Induced Premature Senescence in p53-Deficient Human CellsRazmik Mirzayans, Bonnie Andrais, Gavin Hansen, et al.International Journal of Molecular Sciences|May 18, 2016
The Growing Complexity of Cancer Cell Response to DNA-Damaging Agents: Caspase 3 Mediates Cell Death or Survival?Razmik Mirzayans, Bonnie Andrais, Piyush Kumar, et al.International Journal of Molecular Sciences|April 29, 2017
Significance of Wild-Type p53 Signaling in Suppressing Apoptosis in Response to Chemical Genotoxic Agents: Impact on Chemotherapy OutcomeRazmik Mirzayans, Bonnie Andrais, Piyush Kumar, et al.Journal of Biomedicine & Biotechnology|August 23, 2012
New insights into p53 signaling and cancer cell response to DNA damage: implications for cancer therapyRazmik Mirzayans, Bonnie Andrais, April Scott, et al.Journal of Cellular Physiology|September 27, 2007
Ultraviolet light exposure triggers nuclear accumulation of p21(WAF1) and accelerated senescence in human normal and nucleotide excision repair-deficient fibroblast strainsRazmik Mirzayans, April Scott, Bonnie Andrais, et al.Pageof 30