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Current Environmental Health Reports|February 8, 2018
Long-Term Health Effects and Underlying Biological Mechanisms of Developmental Exposure to ArsenicLisa Smeester, Rebecca C FryReviews on Environmental Health|February 21, 2014
Long-term health consequences of prenatal arsenic exposure: links to the genome and the epigenomeKathryn Bailey, Rebecca C FryEnvironmental Epigenetics|April 12, 2016
A cross-study analysis of prenatal exposures to environmental contaminants and the epigenome: support for stress-responsive transcription factor occupancy as a mediator of gene-specific CpG methylation patterningElizabeth M Martin, Rebecca C FryAnnals of Global Health|June 22, 2016
A Systems Toxicology-based Approach Reveals Biological Pathways Dysregulated by Prenatal Arsenic ExposureJessica E Laine, Rebecca C FryEnvironmental Epigenetics|April 27, 2026
Epigenetic mechanisms linking the perinatal environment, the placenta, and maternal and child health outcomes: evidence for sexual dimorphismHadley J Hartwell, Rebecca C FryFood and Chemical Toxicology : an International Journal Published for the British Industrial Biological Research Association|August 5, 2017
Cadmium inhibits placental trophoblast cell migration via miRNA regulation of the transforming growth factor beta (TGF-β) pathwaySamira A Brooks, Rebecca C FryAnnual Review of Public Health|January 13, 2018
Environmental Influences on the Epigenome: Exposure- Associated DNA Methylation in Human PopulationsElizabeth M Martin, Rebecca C FryCurrent Environmental Health Reports|May 27, 2014
Arsenic-Associated Changes to the Epigenome: What Are the Functional Consequences?Kathryn A Bailey, Rebecca C FryInternational Journal of Environmental Research and Public Health|July 4, 2012
The aryl hydrocarbon receptor pathway: a key component of the microRNA-mediated AML signalisomeJulia E Rager, Rebecca C FryCurrent Environmental Health Reports|August 20, 2020
Perfluoroalkyl Substances (PFAS) and Their Effects on the Placenta, Pregnancy, and Child Development: a Potential Mechanistic Role for Placental Peroxisome Proliferator-Activated Receptors (PPARs)John T Szilagyi, Vennela Avula, Rebecca C FryPageof 30