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Trends in Neurosciences|July 10, 2019
Refueling the Ischemic CNS: Guidance Molecules for Vascular RepairRuslan Rust, Lisa Grönnert, Rebecca Zoe Weber, et al.Expert Opinion on Drug Delivery|March 17, 2025
The blood-brain barrier as a treatment target for neurodegenerative disordersRuslan Rust, Abhay P Sagare, Mingzi Zhang, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 2, 2006
Tau aggregation and progressive neuronal degeneration in the absence of changes in spine density and morphology after targeted expression of Alzheimer's disease-relevant tau constructs in organotypic hippocampal slicesNeelam Shahani, Srinivasa Subramaniam, Tobias Wolf, et al.Brain : a Journal of Neurology|May 18, 2022
Stimulation of the cuneiform nucleus enables training and boosts recovery after spinal cord injuryAnna-Sophie Hofer, Myriam I Scheuber, Andrea M Sartori, et al.Methods in Molecular Biology (Clifton, N.J.)|February 28, 2012
High-resolution imaging and evaluation of spines in organotypic hippocampal slice culturesFrederik Sündermann, Nataliya Golovyashkina, Christian Tackenberg, et al.Brain Pathology (Zurich, Switzerland)|September 1, 2020
Distinct changes in all major components of the neurovascular unit across different neuropathological stages of Alzheimer's diseaseTunahan Kirabali, Ruslan Rust, Serena Rigotti, et al.Frontiers in Immunology|January 9, 2019
T Lymphocytes Contribute to the Control of Baseline Neural Precursor Cell Proliferation but Not the Exercise-Induced Up-Regulation of Adult Hippocampal NeurogenesisTara L Walker, Sonja Schallenberg, Nicole Rund, et al.Translational Psychiatry|August 25, 2025
Fueling the brain - the role of apolipoprotein E in brain energy metabolism and its implications for Alzheimer's diseaseVanessa Budny, Iván Ruminot, Maha Wybitul, et al.Experimental Neurology|November 26, 2021
Slow development of bladder malfunction parallels spinal cord fiber sprouting and interneurons' loss after spinal cord transectionAndrea M Sartori, Anna-Sophie Hofer, Myriam I Scheuber, et al.Plos One|July 25, 2018
Loss of Nogo-A, encoded by the schizophrenia risk gene Rtn4, reduces mGlu3 expression and causes hyperexcitability in hippocampal CA3 circuitsStewart Berry, Oliver Weinmann, Ann-Kristina Fritz, et al.Pageof 8