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Rei J Abe

Showing results (1-10 of 9) with videos related to

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Free Radical Biology & Medicine|October 7, 2021
Disturbed flow-induced FAK K152 SUMOylation initiates the formation of pro-inflammation positive feedback loop by inducing reactive oxygen species production in endothelial cellsLoka Reddy Velatooru, Rei J Abe, Masaki Imanishi, et al.
Frontiers in Cardiovascular Medicine|September 22, 2018
Ponatinib Activates an Inflammatory Response in Endothelial Cells via ERK5 SUMOylationJesus Paez-Mayorga, Andrew L Chen, Sivareddy Kotla, et al.
Current Atherosclerosis Reports|March 25, 2022
Free Cholesterol Bioavailability and AtherosclerosisRei J Abe, Jun-Ichi Abe, Minh T H Nguyen, et al.
Frontiers in Cardiovascular Medicine|November 8, 2021
Senescence-Associated Secretory Phenotype as a Hinge Between Cardiovascular Diseases and CancerPriyanka Banerjee, Sivareddy Kotla, Loka Reddy Velatooru, et al.
Frontiers in Cardiovascular Medicine|March 8, 2021
Corrigendum: p90RSK-MAGI1 Module Controls Endothelial Permeability by Post-translational Modifications of MAGI1 and Hippo PathwayRei J Abe, Hannah Savage, Masaki Imanishi, et al.
Frontiers in Cardiovascular Medicine|December 11, 2020
p90RSK-MAGI1 Module Controls Endothelial Permeability by Post-translational Modifications of MAGI1 and Hippo PathwayRei J Abe, Hannah Savage, Masaki Imanishi, et al.
JCI Insight|April 5, 2019
MAGI1 as a link between endothelial activation and ER stress drives atherosclerosisJun-Ichi Abe, Kyung Ae Ko, Sivareddy Kotla, et al.
Frontiers in Cardiovascular Medicine|September 15, 2023
Endothelial activation and fibrotic changes are impeded by laminar flow-induced CHK1-SENP2 activity through mechanisms distinct from endothelial-to-mesenchymal cell transitionMinh T H Nguyen, Masaki Imanishi, Shengyu Li, et al.
Circulation Research|June 2, 2023
An ERK5-NRF2 Axis Mediates Senescence-Associated Stemness and AtherosclerosisJun-Ichi Abe, Masaki Imanishi, Shengyu Li, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
Free Radical Biology & Medicine|October 7, 2021
Disturbed flow-induced FAK K152 SUMOylation initiates the formation of pro-inflammation positive feedback loop by inducing reactive oxygen species production in endothelial cellsLoka Reddy Velatooru, Rei J Abe, Masaki Imanishi, et al.
Frontiers in Cardiovascular Medicine|September 22, 2018
Ponatinib Activates an Inflammatory Response in Endothelial Cells via ERK5 SUMOylationJesus Paez-Mayorga, Andrew L Chen, Sivareddy Kotla, et al.
Current Atherosclerosis Reports|March 25, 2022
Free Cholesterol Bioavailability and AtherosclerosisRei J Abe, Jun-Ichi Abe, Minh T H Nguyen, et al.
Frontiers in Cardiovascular Medicine|November 8, 2021
Senescence-Associated Secretory Phenotype as a Hinge Between Cardiovascular Diseases and CancerPriyanka Banerjee, Sivareddy Kotla, Loka Reddy Velatooru, et al.
Frontiers in Cardiovascular Medicine|March 8, 2021
Corrigendum: p90RSK-MAGI1 Module Controls Endothelial Permeability by Post-translational Modifications of MAGI1 and Hippo PathwayRei J Abe, Hannah Savage, Masaki Imanishi, et al.
Frontiers in Cardiovascular Medicine|December 11, 2020
p90RSK-MAGI1 Module Controls Endothelial Permeability by Post-translational Modifications of MAGI1 and Hippo PathwayRei J Abe, Hannah Savage, Masaki Imanishi, et al.
JCI Insight|April 5, 2019
MAGI1 as a link between endothelial activation and ER stress drives atherosclerosisJun-Ichi Abe, Kyung Ae Ko, Sivareddy Kotla, et al.
Frontiers in Cardiovascular Medicine|September 15, 2023
Endothelial activation and fibrotic changes are impeded by laminar flow-induced CHK1-SENP2 activity through mechanisms distinct from endothelial-to-mesenchymal cell transitionMinh T H Nguyen, Masaki Imanishi, Shengyu Li, et al.
Circulation Research|June 2, 2023
An ERK5-NRF2 Axis Mediates Senescence-Associated Stemness and AtherosclerosisJun-Ichi Abe, Masaki Imanishi, Shengyu Li, et al.
Pageof 1