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Remus M Berretta

Showing results (21-30 of 42) with videos related to

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Circulation Research|June 27, 2013
Bone-derived stem cells repair the heart after myocardial infarction through transdifferentiation and paracrine signaling mechanismsJason M Duran, Catherine A Makarewich, Thomas E Sharp, et al.
The Journal of Trauma and Acute Care Surgery|January 21, 2017
Acute right heart failure after hemorrhagic shock and trauma pneumonectomy-a management approach: A blinded randomized controlled animal trial using inhaled nitric oxideAndrea L Lubitz, Lars O Sjoholm, Amy Goldberg, et al.
Circulation Research|October 17, 2015
Unique Features of Cortical Bone Stem Cells Associated With Repair of the Injured HeartSadia Mohsin, Constantine D Troupes, Timothy Starosta, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 4, 2023
Effects of maternal hypothyroidism on postnatal cardiomyocyte proliferation and cardiac disease responses of the progenyYijia Li, Jaslyn P Johnson, Yijun Yang, et al.
Circulation Research|February 4, 2012
Hyperphosphorylation of the cardiac ryanodine receptor at serine 2808 is not involved in cardiac dysfunction after myocardial infarctionHongyu Zhang, Catherine A Makarewich, Hajime Kubo, et al.
JACC. Basic to Translational Science|September 1, 2021
Molecular Signature of HFpEF: Systems Biology in a Cardiac-Centric Large Animal ModelAndrew A Gibb, Emma K Murray, Deborah M Eaton, et al.
JACC. Basic to Translational Science|August 1, 2018
Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection FractionThomas E Sharp, Hajime Kubo, Remus M Berretta, et al.
American Journal of Physiology. Heart and Circulatory Physiology|February 10, 2023
Combining three independent pathological stressors induces a heart failure with preserved ejection fraction phenotypeYijia Li, Hajime Kubo, Daohai Yu, et al.
Journal of Molecular and Cellular Cardiology|November 30, 2010
Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophyXiongwen Chen, Hiroyuki Nakayama, Xiaoying Zhang, et al.
Circulation Research|July 23, 2014
Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarctionCatherine A Makarewich, Hongyu Zhang, Jennifer Davis, et al.
Pageof 5

Showing results (21-30 of 42) with videos related to

Sort By:
Pageof 5
Circulation Research|June 27, 2013
Bone-derived stem cells repair the heart after myocardial infarction through transdifferentiation and paracrine signaling mechanismsJason M Duran, Catherine A Makarewich, Thomas E Sharp, et al.
The Journal of Trauma and Acute Care Surgery|January 21, 2017
Acute right heart failure after hemorrhagic shock and trauma pneumonectomy-a management approach: A blinded randomized controlled animal trial using inhaled nitric oxideAndrea L Lubitz, Lars O Sjoholm, Amy Goldberg, et al.
Circulation Research|October 17, 2015
Unique Features of Cortical Bone Stem Cells Associated With Repair of the Injured HeartSadia Mohsin, Constantine D Troupes, Timothy Starosta, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 4, 2023
Effects of maternal hypothyroidism on postnatal cardiomyocyte proliferation and cardiac disease responses of the progenyYijia Li, Jaslyn P Johnson, Yijun Yang, et al.
Circulation Research|February 4, 2012
Hyperphosphorylation of the cardiac ryanodine receptor at serine 2808 is not involved in cardiac dysfunction after myocardial infarctionHongyu Zhang, Catherine A Makarewich, Hajime Kubo, et al.
JACC. Basic to Translational Science|September 1, 2021
Molecular Signature of HFpEF: Systems Biology in a Cardiac-Centric Large Animal ModelAndrew A Gibb, Emma K Murray, Deborah M Eaton, et al.
JACC. Basic to Translational Science|August 1, 2018
Protein Kinase C Inhibition With Ruboxistaurin Increases Contractility and Reduces Heart Size in a Swine Model of Heart Failure With Reduced Ejection FractionThomas E Sharp, Hajime Kubo, Remus M Berretta, et al.
American Journal of Physiology. Heart and Circulatory Physiology|February 10, 2023
Combining three independent pathological stressors induces a heart failure with preserved ejection fraction phenotypeYijia Li, Hajime Kubo, Daohai Yu, et al.
Journal of Molecular and Cellular Cardiology|November 30, 2010
Calcium influx through Cav1.2 is a proximal signal for pathological cardiomyocyte hypertrophyXiongwen Chen, Hiroyuki Nakayama, Xiaoying Zhang, et al.
Circulation Research|July 23, 2014
Transient receptor potential channels contribute to pathological structural and functional remodeling after myocardial infarctionCatherine A Makarewich, Hongyu Zhang, Jennifer Davis, et al.
Pageof 5