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Pflugers Archiv : European Journal of Physiology|June 19, 2012
Mitochondrial Ca²⁺ homeostasis: mechanism, role, and tissue specificitiesPaola Pizzo, Ilaria Drago, Riccardo Filadi, et al.Trends in Neurosciences|February 20, 2021
Excitotoxicity Revisited: Mitochondria on the Verge of a Nervous BreakdownNicoletta Plotegher, Riccardo Filadi, Paola Pizzo, et al.Cell Calcium|May 16, 2023
Mitochondrial Ca2+ signaling and Alzheimer's disease: Too much or too little?Paloma Garcia-Casas, Michela Rossini, Riccardo Filadi, et al.Autophagy|March 21, 2019
PSEN2 (presenilin 2) mutants linked to familial Alzheimer disease impair autophagy by altering Ca2+ homeostasisChiara Fedeli, Riccardo Filadi, Alice Rossi, et al.Proceedings of the National Academy of Sciences of the United States of America|April 15, 2015
Mitofusin 2 ablation increases endoplasmic reticulum-mitochondria couplingRiccardo Filadi, Elisa Greotti, Gabriele Turacchio, et al.Cell Reports|May 31, 2016
Presenilin 2 Modulates Endoplasmic Reticulum-Mitochondria Coupling by Tuning the Antagonistic Effect of Mitofusin 2Riccardo Filadi, Elisa Greotti, Gabriele Turacchio, et al.Cell Reports|February 21, 2020
Defective Mitochondrial Pyruvate Flux Affects Cell Bioenergetics in Alzheimer's Disease-Related ModelsAlice Rossi, Giulia Rigotto, Giulia Valente, et al.Journal of Cellular and Molecular Medicine|May 21, 2016
Mitofusin-2 knockdown increases ER-mitochondria contact and decreases amyloid β-peptide productionNuno Santos Leal, Bernadette Schreiner, Catarina Moreira Pinho, et al.Cell Calcium|December 14, 2020
The yin and yang of mitochondrial Ca2+ signaling in cell physiology and pathologyRiccardo Filadi, Elisa GreottiCell Death and Differentiation|December 13, 2017
SPLICS: a split green fluorescent protein-based contact site sensor for narrow and wide heterotypic organelle juxtapositionDomenico Cieri, Mattia Vicario, Marta Giacomello, et al.Pageof 10