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Richard Fishel

Showing results (71-80 of 96) with videos related to

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EMBO Reports|September 26, 2009
RelA/p65 functions to maintain cellular senescence by regulating genomic stability and DNA repairJingxin Wang, Naduparambil K Jacob, Katherine J Ladner, et al.
The Journal of Biological Chemistry|September 23, 2011
Human MSH2 (hMSH2) protein controls ATP processing by hMSH2-hMSH6Christopher D Heinen, Jennifer L Cyr, Christopher Cook, et al.
Plos One|March 31, 2011
The base excision repair pathway is required for efficient lentivirus integrationKristine E Yoder, Amy Espeseth, Xiao-hong Wang, et al.
Cancer Research|February 28, 2002
Exclusion of breast cancer as an integral tumor of hereditary nonpolyposis colorectal cancerAnnegret Müller, Tina Bocker Edmonston, Diana A Corao, et al.
The Journal of Biological Chemistry|June 13, 2009
Acetylation of histone H3 at the nucleosome dyad alters DNA-histone bindingMridula Manohar, Alex M Mooney, Justin A North, et al.
Carcinogenesis|June 29, 2012
The hMSH2(M688R) Lynch syndrome mutation may function as a dominant negativeJuana V Martín-López, Ysamar Barrios, Vicente Medina-Arana, et al.
Cancer Research|May 6, 2004
Deficiencies in mouse Myh and Ogg1 result in tumor predisposition and G to T mutations in codon 12 of the K-ras oncogene in lung tumorsYali Xie, Hanjing Yang, Cristina Cunanan, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 8, 2014
Mismatch repair protein hMSH2-hMSH6 recognizes mismatches and forms sliding clamps within a D-loop recombination intermediateMasayoshi Honda, Yusuke Okuno, Sarah R Hengel, et al.
The Journal of Biological Chemistry|March 21, 2021
Retroviral prototype foamy virus intasome binding to a nucleosome target does not determine integration efficiencyRandi M Kotlar, Nathan D Jones, Gayan Senavirathne, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 25, 2003
The BCSC-1 locus at chromosome 11q23-q24 is a candidate tumor suppressor geneEric S Martin, Rossano Cesari, Francesca Pentimalli, et al.
Pageof 10

Showing results (71-80 of 96) with videos related to

Sort By:
Pageof 10
EMBO Reports|September 26, 2009
RelA/p65 functions to maintain cellular senescence by regulating genomic stability and DNA repairJingxin Wang, Naduparambil K Jacob, Katherine J Ladner, et al.
The Journal of Biological Chemistry|September 23, 2011
Human MSH2 (hMSH2) protein controls ATP processing by hMSH2-hMSH6Christopher D Heinen, Jennifer L Cyr, Christopher Cook, et al.
Plos One|March 31, 2011
The base excision repair pathway is required for efficient lentivirus integrationKristine E Yoder, Amy Espeseth, Xiao-hong Wang, et al.
Cancer Research|February 28, 2002
Exclusion of breast cancer as an integral tumor of hereditary nonpolyposis colorectal cancerAnnegret Müller, Tina Bocker Edmonston, Diana A Corao, et al.
The Journal of Biological Chemistry|June 13, 2009
Acetylation of histone H3 at the nucleosome dyad alters DNA-histone bindingMridula Manohar, Alex M Mooney, Justin A North, et al.
Carcinogenesis|June 29, 2012
The hMSH2(M688R) Lynch syndrome mutation may function as a dominant negativeJuana V Martín-López, Ysamar Barrios, Vicente Medina-Arana, et al.
Cancer Research|May 6, 2004
Deficiencies in mouse Myh and Ogg1 result in tumor predisposition and G to T mutations in codon 12 of the K-ras oncogene in lung tumorsYali Xie, Hanjing Yang, Cristina Cunanan, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 8, 2014
Mismatch repair protein hMSH2-hMSH6 recognizes mismatches and forms sliding clamps within a D-loop recombination intermediateMasayoshi Honda, Yusuke Okuno, Sarah R Hengel, et al.
The Journal of Biological Chemistry|March 21, 2021
Retroviral prototype foamy virus intasome binding to a nucleosome target does not determine integration efficiencyRandi M Kotlar, Nathan D Jones, Gayan Senavirathne, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 25, 2003
The BCSC-1 locus at chromosome 11q23-q24 is a candidate tumor suppressor geneEric S Martin, Rossano Cesari, Francesca Pentimalli, et al.
Pageof 10