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Methods in Molecular Biology (Clifton, N.J.)|August 16, 2012
Diabetes in Zucker diabetic fatty ratMasakazu Shiota, Richard L Printz
American Journal of Physiology. Endocrinology and Metabolism|January 24, 2024
The impact of glucagon to support postabsorptive glucose flux and glycemia in healthy rats and its attenuation in male Zucker diabetic fatty ratsShanea K Estes, Chiyo Shiota, Tracy P O'Brien, et al.
International Journal of Molecular Sciences|March 28, 2024
Probing Liver Injuries Induced by Thioacetamide in Human In Vitro Pooled Hepatocyte ExperimentsHimanshu Goel, Richard L Printz, Venkat R Pannala, et al.
Frontiers in Pharmacology|November 22, 2018
Identification of the Toxicity Pathways Associated With Thioacetamide-Induced Injuries in Rat Liver and KidneyPatric Schyman, Richard L Printz, Shanea K Estes, et al.
Frontiers in Genetics|December 19, 2019
Assessing Chemical-Induced Liver Injury In Vivo From In Vitro Gene Expression Data in the Rat: The Case of Thioacetamide ToxicityPatric Schyman, Richard L Printz, Shanea K Estes, et al.
International Journal of Molecular Sciences|June 10, 2020
Concordance between Thioacetamide-Induced Liver Injury in Rat and Human In Vitro Gene Expression DataPatric Schyman, Richard L Printz, Shanea K Estes, et al.
Toxicology|June 30, 2020
A toxicogenomic approach to assess kidney injury induced by mercuric chloride in ratsPatric Schyman, Richard L Printz, Mohamed Diwan M AbdulHameed, et al.
American Journal of Physiology. Endocrinology and Metabolism|October 18, 2012
Central nervous system neuropeptide Y signaling via the Y1 receptor partially dissociates feeding behavior from lipoprotein metabolism in lean ratsJennifer M Rojas, John M Stafford, Sanaz Saadat, et al.
Toxicology in Vitro : an International Journal Published in Association with BIBRA|February 3, 2018
Genome-wide gene expression changes associated with exposure of rat liver, heart, and kidney cells to endosulfanRuifeng Liu, Richard L Printz, Erin C Jenkins, et al.
Diabetes|July 21, 2011
Defective glycogenesis contributes toward the inability to suppress hepatic glucose production in response to hyperglycemia and hyperinsulinemia in zucker diabetic fatty ratsTracy P Torres, Yuka Fujimoto, E P Donahue, et al.
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