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Richard T Mathias

Showing results (31-40 of 53) with videos related to

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Biochemical and Biophysical Research Communications|September 11, 2007
Two-pore K+ channels, NO and metabolic inhibitionZhongju Lu, Junyuan Gao, Joan Zuckerman, et al.
The Journal of General Physiology|June 1, 2011
Lens intracellular hydrostatic pressure is generated by the circulation of sodium and modulated by gap junction couplingJunyuan Gao, Xiurong Sun, Leon C Moore, et al.
Investigative Ophthalmology & Visual Science|December 3, 2017
Aquaporin 0 Modulates Lens Gap Junctions in the Presence of Lens-Specific Beaded Filament ProteinsSindhu Kumari, Junyuan Gao, Richard T Mathias, et al.
Investigative Ophthalmology & Visual Science|September 29, 2004
Lens gap junctional coupling is modulated by connexin identity and the locus of gene expressionFrancisco J Martinez-Wittinghan, Caterina Sellitto, Thomas W White, et al.
Investigative Ophthalmology & Visual Science|October 23, 2019
The Ciliary Muscle and Zonules of Zinn Modulate Lens Intracellular Hydrostatic Pressure Through Transient Receptor Potential Vanilloid ChannelsYadi Chen, Junyuan Gao, Leping Li, et al.
Investigative Ophthalmology & Visual Science|June 30, 2020
Signaling Between TRPV1/TRPV4 and Intracellular Hydrostatic Pressure in the Mouse LensNicholas A Delamere, Mohammad Shahidullah, Richard T Mathias, et al.
The Journal of Clinical Investigation|November 26, 2013
AKT activation promotes PTEN hamartoma tumor syndrome-associated cataract developmentCaterina Sellitto, Leping Li, Junyuan Gao, et al.
Biochemical and Biophysical Research Communications|May 12, 2015
Role of Aquaporin 0 in lens biomechanicsS Sindhu Kumari, Neha Gupta, Alan Shiels, et al.
Circulation|September 13, 2003
Both metabolic inhibition and mitochondrial K(ATP) channel opening are myoprotective and initiate a compensatory sarcolemmal outward membrane currentHiroshi Irie, Junyuan Gao, Glenn R Gaudette, et al.
The Journal of Cell Biology|June 5, 2003
Dominant cataracts result from incongruous mixing of wild-type lens connexinsFrancisco J Martinez-Wittinghan, Caterina Sellitto, Leping Li, et al.
Pageof 6

Showing results (31-40 of 53) with videos related to

Sort By:
Pageof 6
Biochemical and Biophysical Research Communications|September 11, 2007
Two-pore K+ channels, NO and metabolic inhibitionZhongju Lu, Junyuan Gao, Joan Zuckerman, et al.
The Journal of General Physiology|June 1, 2011
Lens intracellular hydrostatic pressure is generated by the circulation of sodium and modulated by gap junction couplingJunyuan Gao, Xiurong Sun, Leon C Moore, et al.
Investigative Ophthalmology & Visual Science|December 3, 2017
Aquaporin 0 Modulates Lens Gap Junctions in the Presence of Lens-Specific Beaded Filament ProteinsSindhu Kumari, Junyuan Gao, Richard T Mathias, et al.
Investigative Ophthalmology & Visual Science|September 29, 2004
Lens gap junctional coupling is modulated by connexin identity and the locus of gene expressionFrancisco J Martinez-Wittinghan, Caterina Sellitto, Thomas W White, et al.
Investigative Ophthalmology & Visual Science|October 23, 2019
The Ciliary Muscle and Zonules of Zinn Modulate Lens Intracellular Hydrostatic Pressure Through Transient Receptor Potential Vanilloid ChannelsYadi Chen, Junyuan Gao, Leping Li, et al.
Investigative Ophthalmology & Visual Science|June 30, 2020
Signaling Between TRPV1/TRPV4 and Intracellular Hydrostatic Pressure in the Mouse LensNicholas A Delamere, Mohammad Shahidullah, Richard T Mathias, et al.
The Journal of Clinical Investigation|November 26, 2013
AKT activation promotes PTEN hamartoma tumor syndrome-associated cataract developmentCaterina Sellitto, Leping Li, Junyuan Gao, et al.
Biochemical and Biophysical Research Communications|May 12, 2015
Role of Aquaporin 0 in lens biomechanicsS Sindhu Kumari, Neha Gupta, Alan Shiels, et al.
Circulation|September 13, 2003
Both metabolic inhibition and mitochondrial K(ATP) channel opening are myoprotective and initiate a compensatory sarcolemmal outward membrane currentHiroshi Irie, Junyuan Gao, Glenn R Gaudette, et al.
The Journal of Cell Biology|June 5, 2003
Dominant cataracts result from incongruous mixing of wild-type lens connexinsFrancisco J Martinez-Wittinghan, Caterina Sellitto, Leping Li, et al.
Pageof 6