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Progress in Biophysics and Molecular Biology|April 9, 2009
hERG1 channel activators: a new anti-arrhythmic principleMorten Grunnet, Rie Schultz Hansen, Søren-Peter Olesen
The Journal of Pharmacology and Experimental Therapeutics|February 28, 2007
Pharmacological activation of rapid delayed rectifier potassium current suppresses bradycardia-induced triggered activity in the isolated guinea pig heartRie Schultz Hansen, Søren-Peter Olesen, Morten Grunnet
Naunyn-Schmiedeberg'S Archives of Pharmacology|February 12, 2013
The antiarrhythmic dipeptide ZP1609 (danegaptide) when given at reperfusion reduces myocardial infarct size in pigsAndreas Skyschally, Barbara Walter, Rie Schultz Hansen, et al.
Cellular Physiology and Biochemistry : International Journal of Experimental Cellular Physiology, Biochemistry, and Pharmacology|December 18, 2008
Biophysical characterization of the short QT mutation hERG-N588K reveals a mixed gain-and loss-of-functionMorten Grunnet, Thomas G Diness, Rie Schultz Hansen, et al.
Biochemical and Biophysical Research Communications|April 4, 2006
Frequency-dependent modulation of KCNQ1 and HERG1 potassium channelsThomas Goldin Diness, Rie Schultz Hansen, Søren-Peter Olesen, et al.
Journal of Cardiovascular Pharmacology|July 3, 2008
In vivo effects of the IKr agonist NS3623 on cardiac electrophysiology of the guinea pigRie Schultz Hansen, Søren-Peter Olesen, Lars Christian B Rønn, et al.
Journal of Cardiovascular Pharmacology|March 12, 2011
The duration of pacing-induced atrial fibrillation is reduced in vivo by inhibition of small conductance Ca(2+)-activated K(+) channelsLasse Skibsbye, Jonas Goldin Diness, Ulrik Svane Sørensen, et al.
Heart Rhythm|January 6, 2009
Antiarrhythmic effect of IKr activation in a cellular model of LQT3Jonas Goldin Diness, Rie Schultz Hansen, Jakob Dahl Nissen, et al.
Basic & Clinical Pharmacology & Toxicology|April 24, 2007
Modulation of ERG channels by XE991Pernille Elmedyb, Kirstine Calloe, Nicole Schmitt, et al.
Pflugers Archiv : European Journal of Physiology|September 3, 2008
Activation of big conductance Ca(2+)-activated K (+) channels (BK) protects the heart against ischemia-reperfusion injuryBo Hjorth Bentzen, Oleg Osadchii, Thomas Jespersen, et al.
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