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Immunity|May 24, 2011
Transcription factor Smad-independent T helper 17 cell induction by transforming-growth factor-β is mediated by suppression of eomesoderminKenji Ichiyama, Takashi Sekiya, Naoko Inoue, et al.Molecular Human Reproduction|October 7, 2025
Prophylactic treatment with progesterone decreases murine miscarriage by suppressing the immunostimulatory activity of macrophagesYuki Kaito, Hajime Ino, Yumi Horii, et al.International Immunology|October 24, 2013
Aryl hydrocarbon receptor plays protective roles in ConA-induced hepatic injury by both suppressing IFN-γ expression and inducing IL-22Hiromi Abe, Akihiro Kimura, Sanae Tsuruta, et al.Cancer Science|May 24, 2018
Generation and application of human induced-stem cell memory T cells for adoptive immunotherapyTaisuke Kondo, Yuki Imura, Shunsuke Chikuma, et al.Cancer Letters|June 1, 2011
Suppression of SOCS3 in macrophages prevents cancer metastasis by modifying macrophage phase and MCP2/CCL8 inductionKiyokazu Hiwatashi, Taiga Tamiya, Eiichi Hasegawa, et al.Arthritis Research & Therapy|May 16, 2016
Identification of definitive serum biomarkers associated with disease activity in primary Sjögren's syndromeAyumi Nishikawa, Katsuya Suzuki, Yoshiaki Kassai, et al.Scientific Reports|August 21, 2025
Inflammatory cytokine interleukin-18 prevents murine miscarriage by inducing appropriate inflammationYumi Horii, Hajime Ino, Fumiyuki Sasaki, et al.Plos One|September 24, 2013
TRAF6 is essential for maintenance of regulatory T cells that suppress Th2 type autoimmunityGo Muto, Hitoshi Kotani, Taisuke Kondo, et al.The Journal of Biological Chemistry|August 25, 2011
Histone 3 lysine 9 (H3K9) methyltransferase recruitment to the interleukin-2 (IL-2) promoter is a mechanism of suppression of IL-2 transcription by the transforming growth factor-β-Smad pathwayYu Wakabayashi, Taiga Tamiya, Ichiro Takada, et al.Arthritis Research & Therapy|July 15, 2016
Enhanced IgG4 production by follicular helper 2 T cells and the involvement of follicular helper 1 T cells in the pathogenesis of IgG4-related diseaseMitsuhiro Akiyama, Hidekata Yasuoka, Kunihiro Yamaoka, et al.Pageof 9