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Genes to Cells : Devoted to Molecular & Cellular Mechanisms|May 12, 2026
Murine Embryonic Stem Cells Possess Distinct E2F Activity That Activates Tumor Suppressor Promoter ElementsYaxuan Zhou, Rinka Nakajima, Mashiro Shirasawa, et al.Cells|December 24, 2025
Utility of Tumor Suppressor E2F Target Gene Promoter Elements to Drive Gene Expression Specifically in Cancer CellsKenta Kurayoshi, Masakazu Tanaka, Rinka Nakajima, et al.Molecular Cancer Therapeutics|January 24, 2026
Chromobox2 inhibition: a novel activity of alisertib, an aurora A kinase inhibitorTomomi M Yamamoto, Ritsuko Iwanaga, Elizabeth R Woodruff, et al.Genes|February 25, 2023
Deregulated E2F Activity as a Cancer-Cell Specific Therapeutic ToolRinka Nakajima, Lin Zhao, Yaxuan Zhou, et al.Molecular Carcinogenesis|June 21, 2020
Loss of prdm1a accelerates melanoma onset and progressionRitsuko Iwanaga, Brittany T Truong, Jessica Y Hsu, et al.Breast Cancer Research : BCR|July 7, 2012
Expression of Six1 in luminal breast cancers predicts poor prognosis and promotes increases in tumor initiating cells by activation of extracellular signal-regulated kinase and transforming growth factor-beta signaling pathwaysRitsuko Iwanaga, Chu-An Wang, Douglas S Micalizzi, et al.Biomolecules|November 27, 2024
The N-Terminal Region of the Transcription Factor E2F1 Contains a Novel Transactivation Domain and Recruits General Transcription Factor GTF2H2Lin Zhao, Rinka Nakajima, Yaxuan Zhou, et al.Expert Opinion on Therapeutic Targets|May 27, 2023
Novel chromobox 2 inhibitory peptide decreases tumor progressionLindsay W Brubaker, Donald S Backos, Vu T Nguyen, et al.International Journal of Molecular Sciences|January 8, 2025
DEAD/H Box 5 (DDX5) Augments E2F1-Induced Cell Death Independent of the Tumor Suppressor p53Rinka Nakajima, Yaxuan Zhou, Mashiro Shirasawa, et al.Biochemical and Biophysical Research Communications|May 4, 2023
The TFDP1 gene coding for DP1, the heterodimeric partner of the transcription factor E2F, is a target of deregulated E2FRinka Nakajima, Reika Deguchi, Hideyuki Komori, et al.Pageof 4