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Robert A Roth

Showing results (31-40 of 87) with videos related to

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Toxicological Sciences : an Official Journal of the Society of Toxicology|June 21, 2002
Bacterial lipopolysaccharide exposure alters aflatoxin B(1) hepatotoxicity: benchmark dose analysis for markers of liver injuryJames P Luyendyk, Kate C Shores, Patricia E Ganey, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|January 20, 2011
Natural killer cells mediate severe liver injury in a murine model of halothane hepatitisChristine M Dugan, Aaron M Fullerton, Robert A Roth, et al.
The Journal of Pharmacology and Experimental Therapeutics|February 15, 2014
Trovafloxacin potentiation of lipopolysaccharide-induced tumor necrosis factor release from RAW 264.7 cells requires extracellular signal-regulated kinase and c-Jun N-Terminal KinaseKyle L Poulsen, Ryan P Albee, Patricia E Ganey, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|August 19, 2007
The role of tumor necrosis factor alpha in lipopolysaccharide/ranitidine-induced inflammatory liver injuryFrancis F Tukov, James P Luyendyk, Patricia E Ganey, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|December 6, 2003
15-deoxy prostaglandin J2 enhances allyl alcohol-induced toxicity in rat hepatocytesJane F Maddox, Alison C Domzalski, Robert A Roth, et al.
The Journal of Pharmacology and Experimental Therapeutics|January 24, 2002
Underlying endotoxemia augments toxic responses to chlorpromazine: is there a relationship to drug idiosyncrasy?John P Buchweitz, Patricia E Ganey, Steven J Bursian, et al.
Journal of Toxicology and Environmental Health. Part A|July 23, 2002
The temporal relationship between bacterial lipopolysaccharide and monocrotaline exposures influences toxicity: shift in response from hepatotoxicity to nitric oxide-dependent lethalitySteven B Yee, Bryan L Copple, Patricia E Ganey, et al.
The Journal of Pharmacology and Experimental Therapeutics|February 4, 2010
A mouse model of severe halothane hepatitis based on human risk factorsChristine M Dugan, Allen E MacDonald, Robert A Roth, et al.
Chemico-Biological Interactions|May 30, 2021
Fenofibrate, a peroxisome proliferator-activated receptor-alpha agonist, blocks steatosis and alters the inflammatory response in a mouse model of inflammation-dioxin interactionJesus Olivero-Verbel, Jack R Harkema, Robert A Roth, et al.
Toxicology|October 25, 2011
Neutrophil-cytokine interactions in a rat model of sulindac-induced idiosyncratic liver injuryWei Zou, Robert A Roth, Husam S Younis, et al.
Pageof 9

Showing results (31-40 of 87) with videos related to

Sort By:
Pageof 9
Toxicological Sciences : an Official Journal of the Society of Toxicology|June 21, 2002
Bacterial lipopolysaccharide exposure alters aflatoxin B(1) hepatotoxicity: benchmark dose analysis for markers of liver injuryJames P Luyendyk, Kate C Shores, Patricia E Ganey, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|January 20, 2011
Natural killer cells mediate severe liver injury in a murine model of halothane hepatitisChristine M Dugan, Aaron M Fullerton, Robert A Roth, et al.
The Journal of Pharmacology and Experimental Therapeutics|February 15, 2014
Trovafloxacin potentiation of lipopolysaccharide-induced tumor necrosis factor release from RAW 264.7 cells requires extracellular signal-regulated kinase and c-Jun N-Terminal KinaseKyle L Poulsen, Ryan P Albee, Patricia E Ganey, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|August 19, 2007
The role of tumor necrosis factor alpha in lipopolysaccharide/ranitidine-induced inflammatory liver injuryFrancis F Tukov, James P Luyendyk, Patricia E Ganey, et al.
Toxicological Sciences : an Official Journal of the Society of Toxicology|December 6, 2003
15-deoxy prostaglandin J2 enhances allyl alcohol-induced toxicity in rat hepatocytesJane F Maddox, Alison C Domzalski, Robert A Roth, et al.
The Journal of Pharmacology and Experimental Therapeutics|January 24, 2002
Underlying endotoxemia augments toxic responses to chlorpromazine: is there a relationship to drug idiosyncrasy?John P Buchweitz, Patricia E Ganey, Steven J Bursian, et al.
Journal of Toxicology and Environmental Health. Part A|July 23, 2002
The temporal relationship between bacterial lipopolysaccharide and monocrotaline exposures influences toxicity: shift in response from hepatotoxicity to nitric oxide-dependent lethalitySteven B Yee, Bryan L Copple, Patricia E Ganey, et al.
The Journal of Pharmacology and Experimental Therapeutics|February 4, 2010
A mouse model of severe halothane hepatitis based on human risk factorsChristine M Dugan, Allen E MacDonald, Robert A Roth, et al.
Chemico-Biological Interactions|May 30, 2021
Fenofibrate, a peroxisome proliferator-activated receptor-alpha agonist, blocks steatosis and alters the inflammatory response in a mouse model of inflammation-dioxin interactionJesus Olivero-Verbel, Jack R Harkema, Robert A Roth, et al.
Toxicology|October 25, 2011
Neutrophil-cytokine interactions in a rat model of sulindac-induced idiosyncratic liver injuryWei Zou, Robert A Roth, Husam S Younis, et al.
Pageof 9