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Journal of Toxicology and Environmental Health. Part A|December 3, 2009
Bacterial- and viral-induced inflammation increases sensitivity to acetaminophen hepatotoxicityJane F Maddox, Chidozie J Amuzie, Maoxiang Li, et al.Blood|July 17, 2015
Platelets and protease-activated receptor-4 contribute to acetaminophen-induced liver injury in miceKazuhisa Miyakawa, Nikita Joshi, Bradley P Sullivan, et al.Toxicological Sciences : an Official Journal of the Society of Toxicology|January 18, 2006
Unique gene expression and hepatocellular injury in the lipopolysaccharide-ranitidine drug idiosyncrasy rat model: comparison with famotidineJames P Luyendyk, Lois D Lehman-McKeeman, David M Nelson, et al.Toxicology and Applied Pharmacology|July 30, 2005
Metabonomic evaluation of idiosyncrasy-like liver injury in rats cotreated with ranitidine and lipopolysaccharideJane F Maddox, James P Luyendyk, Gregory N Cosma, et al.Hepatology (Baltimore, Md.)|June 9, 2021
Key Characteristics of Human Hepatotoxicants as a Basis for Identification and Characterization of the Causes of Liver ToxicityIvan Rusyn, Xabier Arzuaga, Russell C Cattley, et al.The Journal of Pharmacology and Experimental Therapeutics|January 13, 2006
Coagulation-dependent gene expression and liver injury in rats given lipopolysaccharide with ranitidine but not with famotidineJames P Luyendyk, Lois D Lehman-McKeeman, David M Nelson, et al.International Journal of Molecular Sciences|December 15, 2010
Comparison of TNFα to lipopolysaccharide as an inflammagen to characterize the idiosyncratic hepatotoxicity potential of drugs: Trovafloxacin as an exampleMichael J Liguori, Amy C Ditewig, Jane F Maddox, et al.Pageof 9