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The Journal of Biological Chemistry|July 10, 2008
CCL2 protects prostate cancer PC3 cells from autophagic death via phosphatidylinositol 3-kinase/AKT-dependent survivin up-regulationHernan Roca, Zachary Varsos, Kenneth J PientaInterface Focus|August 7, 2014
Critical transitions in a game theoretic model of tumour metabolismArdeshir Kianercy, Robert Veltri, Kenneth J PientaOncotarget|May 15, 2018
Epigenetic control of macrophage polarization: implications for targeting tumor-associated macrophagesAmber E de Groot, Kenneth J PientaCancer Metastasis Reviews|January 4, 2023
Dormant cancer cells: programmed quiescence, senescence, or both?Kevin Truskowski, Sarah R Amend, Kenneth J PientaInternational Journal of Molecular Sciences|September 13, 2025
Methods and Guidelines for Metabolism Studies: Applications to Cancer ResearchMelvin Li, Sarah R Amend, Kenneth J PientaCurrent Opinion in Oncology|February 19, 2021
Understanding the tumor-immune microenvironment in prostate cancerLiang Dong, Kayla V Myers, Kenneth J PientaNeoplasia (New York, N.Y.)|December 19, 2009
CCL2 is a negative regulator of AMP-activated protein kinase to sustain mTOR complex-1 activation, survivin expression, and cell survival in human prostate cancer PC3 cellsHernan Roca, Zachary S Varsos, Kenneth J PientaOncology (Williston Park, N.Y.)|December 3, 1999
Pain management in patients with advanced prostate cancerKarin B Olson Pa-C, Kenneth J PientaJournal of Cellular Biochemistry|March 3, 2004
Dynamic process of prostate cancer metastasis to boneKwanchanit Tantivejkul, Linda M Kalikin, Kenneth J PientaArtificial Life|September 7, 2006
Simulating the hallmarks of cancerRobert G Abbott, Stephanie Forrest, Kenneth J PientaPageof 52