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Blood|March 30, 2010
Protein S controls hypoxic/ischemic blood-brain barrier disruption through the TAM receptor Tyro3 and sphingosine 1-phosphate receptorDonghui Zhu, Yaoming Wang, Itender Singh, et al.
Hypertension (Dallas, Tex. : 1979)|May 23, 2012
Hypertension induces brain β-amyloid accumulation, cognitive impairment, and memory deterioration through activation of receptor for advanced glycation end products in brain vasculatureDaniela Carnevale, Giada Mascio, Ivana D'Andrea, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 12, 2007
Serum response factor and myocardin mediate arterial hypercontractility and cerebral blood flow dysregulation in Alzheimer's phenotypeNienwen Chow, Robert D Bell, Rashid Deane, et al.
Translational Psychiatry|February 20, 2021
Schizophrenia-associated SLC39A8 polymorphism is a loss-of-function allele altering glutamate receptor and innate immune signalingWei Chou Tseng, Veronica Reinhart, Thomas A Lanz, et al.
Blood Advances|April 28, 2018
Abnormal clotting of the intrinsic/contact pathway in Alzheimer disease patients is related to cognitive abilityGeorgette L Suidan, Pradeep K Singh, Sunita Patel-Hett, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 19, 2010
Protein S protects neurons from excitotoxic injury by activating the TAM receptor Tyro3-phosphatidylinositol 3-kinase-Akt pathway through its sex hormone-binding globulin-like regionZhihui Zhong, Yaoming Wang, Huang Guo, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|March 1, 2014
Identification and initial functional characterization of a human vascular cell-enriched long noncoding RNARobert D Bell, Xiaochun Long, Mingyan Lin, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 5, 2013
An activated protein C analog stimulates neuronal production by human neural progenitor cells via a PAR1-PAR3-S1PR1-Akt pathwayHuang Guo, Zhen Zhao, Qi Yang, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 21, 2013
Low levels of copper disrupt brain amyloid-β homeostasis by altering its production and clearanceItender Singh, Abhay P Sagare, Mireia Coma, et al.
Journal of Neuroinflammation|May 16, 2018
Inhibition of 2-AG hydrolysis differentially regulates blood brain barrier permeability after injuryJustin R Piro, Georgette L Suidan, Jie Quan, et al.
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