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Neoplasia (New York, N.Y.)|March 15, 2006
Inhibition of decay-accelerating factor (CD55) attenuates prostate cancer growth and survival in vivoRobert D Loberg, LaShon L Day, Rodney Dunn, et al.
Bioinformatics Advances|March 10, 2025
XeroGraph: enhancing data integrity in the presence of missing values with statistical and predictive analysisLaila Mousafi Alasal, Emma U Hammarlund, Kenneth J Pienta, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 5, 2025
Strategic analysis of dissent and self-censorshipJoshua J Daymude, Robert Axelrod, Stephanie Forrest
Proceedings of the National Academy of Sciences of the United States of America|December 8, 2021
Preventing extreme polarization of political attitudesRobert Axelrod, Joshua J Daymude, Stephanie Forrest
The Journal of Biological Chemistry|October 17, 2009
CCL2 and interleukin-6 promote survival of human CD11b+ peripheral blood mononuclear cells and induce M2-type macrophage polarizationHernan Roca, Zachary S Varsos, Sudha Sud, et al.
The Prostate|June 19, 2008
Dickkopf-1 expression increases early in prostate cancer development and decreases during progression from primary tumor to metastasisChristopher L Hall, Stephanie D Daignault, Rajal B Shah, et al.
The Prostate|November 14, 2022
Presence of cells in the polyaneuploid cancer cell (PACC) state predicts the risk of recurrence in prostate cancerLevent Trabzonlu, Kenneth J Pienta, Bruce J Trock, et al.
Pharmacological Research|October 9, 2008
-(-)Gossypol promotes the apoptosis of bladder cancer cells in vitroJill A Macoska, Shreelekha Adsule, Kwanchanit Tantivejkul, et al.
Neoplasia (New York, N.Y.)|August 22, 2007
CCL2 as an important mediator of prostate cancer growth in vivo through the regulation of macrophage infiltrationRobert D Loberg, Chi Ying, Matt Craig, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
Mitotic bypass and endocycling promote cancer cell survival after genotoxic chemotherapyKevin Truskowski, Louis Rolle, George Butler, et al.
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