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The Biochemical Journal|November 1, 2002
Ceramide: second messenger or modulator of membrane structure and dynamics?Wim J van Blitterswijk, Arnold H van der Luit, Robert Jan Veldman, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 1, 2002
Altered sphingolipid metabolism in multidrug-resistant ovarian cancer cells is due to uncoupling of glycolipid biosynthesis in the Golgi apparatusRobert Jan Veldman, Karin Klappe, John Hinrichs, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|September 19, 2002
Ara-C- and daunorubicin-induced recruitment of Lyn in sphingomyelinase-enriched membrane raftsSolène Grazide, Nicolas Maestre, Robert Jan Veldman, et al.
The Journal of Pharmacology and Experimental Therapeutics|July 26, 2005
Coformulated N-octanoyl-glucosylceramide improves cellular delivery and cytotoxicity of liposomal doxorubicinRobert Jan Veldman, Gerben A Koning, Albert van Hell, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|April 15, 2003
The absence of functional glucosylceramide synthase does not sensitize melanoma cells for anticancer drugsRobert Jan Veldman, Alain Mita, Olivier Cuvillier, et al.
Toxicon : Official Journal of the International Society on Toxinology|February 8, 2024
Distinct cardiotoxic effects by venoms of a spitting cobra (Naja pallida) and a rattlesnake (Crotalus atrox) revealed using an ex vivo Langendorff heart modelRonald Vlasblom, Jory van Thiel, Matyas A Bittenbinder, et al.
Nutrients|September 14, 2018
Release of Major Peanut Allergens from Their Matrix under Various pH and Simulated Saliva Conditions-Ara h2 and Ara h6 Are Readily Bio-AccessibleStef J Koppelman, Mieke Smits, Monic Tomassen, et al.
The Journal of Biological Chemistry|April 14, 2004
Accumulation of glycosphingolipids in Niemann-Pick C disease disrupts endosomal transportDanielle te Vruchte, Emyr Lloyd-Evans, Robert Jan Veldman, et al.
The Journal of Gene Medicine|May 12, 2009
AAV gene therapy as a means to increase apolipoprotein (Apo) A-I and high-density lipoprotein-cholesterol levels: correction of murine ApoA-I deficiencyStefan F C Vaessen, Robert Jan Veldman, Elisabeth M Comijn, et al.
The Biochemical Journal|March 9, 2006
ABCA1-dependent but apoA-I-independent cholesterol efflux mediated by fatty acid-bile acid conjugates (FABACs)Ilana Goldiner, Astrid E van der Velde, Kristin E Vandenberghe, et al.
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