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Journal of Immunology (Baltimore, Md. : 1950)|August 22, 2007
CD8+ T cell activation is governed by TCR-peptide/MHC affinity, not dissociation rateShaomin Tian, Robert Maile, Edward J Collins, et al.
Frontiers in Immunology|December 10, 2025
DAMP-driven trained immunity: metabolic and epigenetic reprogramming in critical illness and chronic inflammationHan G Kim, Jaimar C Rincon, Philip A Efron, et al.
Immunology|December 21, 2004
Memory CD8 T cells require CD8 coreceptor engagement for calcium mobilization and proliferation, but not cytokine productionSamantha E Kerry, Robert Maile, Edward J Collins, et al.
International Journal of Burns and Trauma|November 26, 2013
Burn injury induces high levels of phosphorylated insulin-like growth factor binding protein-1April E Mendoza, Laura A Maile, Bruce A Cairns, et al.
The Journal of Trauma|August 19, 2006
Increased Toll-like receptor 4 expression on T cells may be a mechanism for enhanced T cell response late after burn injuryBruce Cairns, Robert Maile, Carie M Barnes, et al.
The Journal of Trauma|April 12, 2008
Toll-like receptor 2 and 4 ligation results in complex altered cytokine profiles early and late after burn injuryBruce A Cairns, Carie M Barnes, Stefan Mlot, et al.
Shock (Augusta, Ga.)|December 14, 2022
EXTRACELLULAR VESICLES AS REGULATORS OF IMMUNE FUNCTION IN TRAUMATIC INJURIES AND SEPSISRoland F Seim, Micah L Willis, Shannon M Wallet, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 10, 2002
Peptidic termini play a significant role in TCR recognitionBo Wang, Ashawni Sharma, Robert Maile, et al.
Plos One|March 31, 2018
HMGB1/IL-1β complexes in plasma microvesicles modulate immune responses to burn injuryLeon G Coleman, Robert Maile, Samuel W Jones, et al.
European Journal of Immunology|January 13, 2006
Low-avidity CD8lo T cells induced by incomplete antigen stimulation in vivo regulate naive higher avidity CD8hi T cell responses to the same antigenRobert Maile, Shannon M Pop, Roland Tisch, et al.
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