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Molecular Neurodegeneration|January 16, 2013
Neuron loss in the 5XFAD mouse model of Alzheimer's disease correlates with intraneuronal Aβ42 accumulation and Caspase-3 activationWilliam A Eimer, Robert VassarThe Journal of Clinical Investigation|July 18, 2023
Connections between ApoE, sleep, and Aβ and tau pathologies in Alzheimer's diseaseKatherine R Sadleir, Robert VassarCurrent Alzheimer Research|June 5, 2014
The normal and pathologic roles of the Alzheimer's β-secretase, BACE1Patty C Kandalepas, Robert VassarJournal of Neuroinflammation|November 4, 2011
The contribution of activated astrocytes to Aβ production: implications for Alzheimer's disease pathogenesisJie Zhao, Tracy O'Connor, Robert VassarThe Journal of Biological Chemistry|October 14, 2018
ER stress is not elevated in the 5XFAD mouse model of Alzheimer's diseaseKatherine R Sadleir, Jelena Popovic, Robert VassarNature Reviews. Neurology|February 9, 2010
The secretases: enzymes with therapeutic potential in Alzheimer diseaseBart De Strooper, Robert Vassar, Todd GoldeThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 25, 2005
Energy inhibition elevates beta-secretase levels and activity and is potentially amyloidogenic in APP transgenic mice: possible early events in Alzheimer's disease pathogenesisRodney A Velliquette, Tracy O'Connor, Robert VassarThe Journal of Biological Chemistry|July 9, 2005
Beta-amyloid-induced dynamin 1 depletion in hippocampal neurons. A potential mechanism for early cognitive decline in Alzheimer diseaseBrent L Kelly, Robert Vassar, Adriana FerreiraJournal of Alzheimer'S Disease Reports|July 24, 2023
Lower Cerebrospinal Fluid Amyloid-β<sub>42</sub> Predicts Sooner Time to Antipsychotic Use in Alzheimer's DiseaseJoshua G Cahan, Robert Vassar, Borna BonakdarpourNeuroscience and Biobehavioral Reviews|April 6, 2016
Inhibiting BACE1 to reverse synaptic dysfunctions in Alzheimer's diseaseRiqiang Yan, Qingyuan Fan, John Zhou, et al.Pageof 12